(Q42536078)

English

Hydrogen peroxide-induced apoptosis is CD95-independent, requires the release of mitochondria-derived reactive oxygen species and the activation of NF-kappaB.

scientific article published on January 1999

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Europe PubMed Central

PubMed publication ID

1 November 2017

Hydrogen peroxide-induced apoptosis is CD95-independent, requires the release of mitochondria-derived reactive oxygen species and the activation of NF-kappaB. (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

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reactive oxygen species

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apoptotic process

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author name string

A Dumont

1

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Europe PubMed Central

PubMed publication ID

1 November 2017

S P Hehner

2

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Europe PubMed Central

PubMed publication ID

1 November 2017

T G Hofmann

3

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

M Ueffing

4

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

W Dröge

5

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

M L Schmitz

6

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

language of work or name

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publication date

1 January 1999

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Europe PubMed Central

PubMed publication ID

1 November 2017

language of work

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Europe PubMed Central

PubMed publication ID

1 November 2017

18

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Europe PubMed Central

PubMed publication ID

1 November 2017

3

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Europe PubMed Central

PubMed publication ID

1 November 2017

747-757

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Europe PubMed Central

PubMed publication ID

1 November 2017

High levels of c-rel expression are associated with programmed cell death in the developing avian embryo and in bone marrow cells in vitro

1 reference

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The NF-kappa B and I kappa B proteins: new discoveries and insights

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Role of reactive oxygen intermediates in activation-induced CD95 (APO-1/Fas) ligand expression

1 reference

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Activation of protein kinase C attenuates early signals in Fas-mediated apoptosis

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Global ischemia activates nuclear factor-kappa B in forebrain neurons of rats

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Role of cysteine and glutathione in HIV infection and other diseases associated with muscle wasting and immunological dysfunction

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Functions of glutathione and glutathione disulfide in immunology and immunopathology.

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New members of the Bcl-2 family and their protein partners

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Distinct effects of glutathione disulphide on the nuclear transcription factor kappa B and the activator protein-1.

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Bcl-2 down-regulates the activity of transcription factor NF-kappaB induced upon apoptosis

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Induction of apoptosis by human Nbk/Bik, a BH3-containing protein that interacts with E1B 19K

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Sesquiterpene lactones specifically inhibit activation of NF-kappa B by preventing the degradation of I kappa B-alpha and I kappa B-beta

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C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2.

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Mediation of c-Myc-induced apoptosis by p53.

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Characterization of hydrogen peroxide toxicity in cultured rat forebrain neurons.

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No requirement of reactive oxygen intermediates in Fas‐mediated apoptosis

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Reactive oxygen intermediates are involved in the induction of CD95 ligand mRNA expression by cytostatic drugs in hepatoma cells

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Differential effects of expression of the CD45 tyrosine protein phosphatase on the tyrosine phosphorylation of the lck, fyn, and c-src tyrosine protein kinases

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Mitogenic signaling mediated by oxidants in Ras-transformed fibroblasts

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BHRF1, a viral homologue of the Bcl-2 oncogene, is conserved at both the sequence and functional level in different Epstein-Barr virus isolates

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Activation of protein kinase C by tyrosine phosphorylation in response to H2O2.

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Mitochondrial control of apoptosis

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A hydrophobic region within the adenovirus E1B 19 kDa protein is necessary for the transient inhibition of NF-kappaB activated by different stimuli.

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Thiol agents and Bcl-2 identify an alphavirus-induced apoptotic pathway that requires activation of the transcription factor NF-kappa B.

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IL-2 gene expression and NF-kappa B activation through CD28 requires reactive oxygen production by 5-lipoxygenase

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Mitochondrial proliferation and paradoxical membrane depolarization during terminal differentiation and apoptosis in a human colon carcinoma cell line

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Mitochondrial permeability transition is a central coordinating event of apoptosis

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H2O2 and antioxidants have opposite effects on activation of NF-kappa B and AP-1 in intact cells: AP-1 as secondary antioxidant-responsive factor.

1 reference

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FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

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Redox regulation of cellular activation

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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A model for p53-induced apoptosis

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Nuclear factor-kappa B contributes to excitotoxin-induced apoptosis in rat striatum

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The adenovirus E1A proteins induce apoptosis, which is inhibited by the E1B 19-kDa and Bcl-2 proteins

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Caspases: intracellular signaling by proteolysis

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Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis

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FLICE is predominantly expressed as two functionally active isoforms, caspase-8/a and caspase-8/b.

1 reference

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Two CD95 (APO-1/Fas) signaling pathways

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The dual effect of adenovirus type 5 E1A 13S protein on NF-kappaB activation is antagonized by E1B 19K.

1 reference

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APO-1(CD95)-mediated apoptosis in Jurkat cells does not involve src kinases or CD45

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Signalling mechanisms and oxidative stress in apoptosis

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Mutational analysis of the transforming and apoptosis suppression activities of the adenovirus E1B 175R protein

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rac1 regulates a cytokine-stimulated, redox-dependent pathway necessary for NF-kappaB activation

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1 reference

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Inhibition of TNF-induced apoptosis by NF-kappa B.

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Caspases and caspase inhibitors

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Advanced glycation end products (AGEs) on the surface of diabetic erythrocytes bind to the vessel wall via a specific receptor inducing oxidant stress in the vasculature: a link between surface-associated AGEs and diabetic complications

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Role of adenovirus E1B proteins in transformation: altered organization of intermediate filaments in transformed cells that express the 19-kilodalton protein

1 reference

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

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Protein-tyrosine phosphorylation regulates apoptosis in human eosinophils and neutrophils.

1 reference

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Mitochondrial control of nuclear apoptosis.

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Identifiers

10.1038/SJ.ONC.1202325

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Dimensions Publication ID

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

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