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2003, Turkish journal of haematology : official journal of Turkish Society of Haematology
We evaluated the prevalence of hepatitis G virus (HGV) infection in patients who had received multiple blood components and in blood donors and the possible coinfection with hepatitis C virus (HCV). Eighty patients who had received multiple blood components and 70 eligible blood donors, as controls were included in this study. HGV RNA was determined by reverse polymerase chain reaction in serum. HGV-RNA was detected in three (3.7%) of patients and in one (1.4%) of controls. Statistical analysis showed no difference between the groups (p> 0.05). HGV and HCV coinfection was not observed in both patient and control groups. Although the most important risk factor for HGV infection was found to be blood transfusions in various studies, this study showed that there is not significant relationship between blood components transfusion and HGV infection.
Hepatitis Monthly, 2009
The Journal of Infectious Diseases, 1999
Transfusion, 1997
New England Journal of Medicine, 1997
Background The role of hepatitis G virus (HGV) in transfusion-associated infection and its relation to liver disease are not well understood. Methods Serum samples collected between 1972 and 1995 from 357 transfusion recipients, 157 con- trols who did not receive transfusions, 500 randomly selected volunteer blood donors, and 230 donors of blood received by HGV-infected patients were tested for HGV
Transfusion, 1999
The Journal of Infection in Developing Countries, 2014
Introduction: Screening blood donors for blood-borne pathogens is very critical for the recipient’s safety. Similar to hepatitis B and C infections, hepatitis G infection is transmitted through contaminated blood and causes acute and chronic hepatitis. Previous reports have shown that the prevalence of hepatitis G virus (HGV) RNA among healthy Saudi donors was 1%-2%. However, the exposure rate of this virus has never been studied. We hypothesized that the prevalence of HGV infection may have changed overtime due to socio-economic and environmental factors. Since hepatitis B and C infections are endemic in Saudi Arabia, we investigated the exposure rate of HGV infection in healthy donors and chronically infected hepatitis B and C patients. Methodology: A prospective study was done on healthy donors and patients with chronic HBV and HCV infections. Hepatitis B and C viral loads were measured by real-time polymerase chain reaction. HGV exposure rate was evaluated by detection of HGV an...
Journal of Viral Hepatitis, 1997
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