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    HCVD Cad Cva Infarction

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    Pamela Domingo
    This document outlines the pathophysiology of coronary artery disease. Non-modifiable risks include heredity, race, and increasing age over 40, while modifiable risks are smoking, hypertension, high cholesterol, and physical inactivity. These risks lead to the deposition of fat on blood vessel walls called atherosclerosis. Coronary artery disease results from stenosis of the coronary arterial wall from atherosclerosis, which decreases blood flow to the heart. Hypertension further increases pressure on blood vessel walls. Plaque dislodgement can also cause emboli that obstruct blood vessels in other parts of the body such as the brain, decreasing blood flow and oxygen supply to brain tissue and resulting in infarction.

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    HCVD Cad Cva Infarction

    Uploaded by

    Pamela Domingo
    98 views2 pages
    This document outlines the pathophysiology of coronary artery disease. Non-modifiable risks include heredity, race, and increasing age over 40, while modifiable risks are smoking, hypertension, high cholesterol, and physical inactivity. These risks lead to the deposition of fat on blood vessel walls called atherosclerosis. Coronary artery disease results from stenosis of the coronary arterial wall from atherosclerosis, which decreases blood flow to the heart. Hypertension further increases pressure on blood vessel walls. Plaque dislodgement can also cause emboli that obstruct blood vessels in other parts of the body such as the brain, decreasing blood flow and oxygen supply to brain tissue and resulting in infarction.

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    108693176 Hcvd Cad Cva Infarction

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    This document outlines the pathophysiology of coronary artery disease. Non-modifiable risks include heredity, race, and increasing age over 40, while modifiable risks are smoking, hypertension, high cholesterol, and physical inactivity. These risks lead to the deposition of fat on blood vessel walls called atherosclerosis. Coronary artery disease results from stenosis of the coronary arterial wall from atherosclerosis, which decreases blood flow to the heart. Hypertension further increases pressure on blood vessel walls. Plaque dislodgement can also cause emboli that obstruct blood vessels in other parts of the body such as the brain, decreasing blood flow and oxygen supply to brain tissue and resulting in infarction.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
    98 views2 pages

    HCVD Cad Cva Infarction

    Uploaded by

    Pamela Domingo
    This document outlines the pathophysiology of coronary artery disease. Non-modifiable risks include heredity, race, and increasing age over 40, while modifiable risks are smoking, hypertension, high cholesterol, and physical inactivity. These risks lead to the deposition of fat on blood vessel walls called atherosclerosis. Coronary artery disease results from stenosis of the coronary arterial wall from atherosclerosis, which decreases blood flow to the heart. Hypertension further increases pressure on blood vessel walls. Plaque dislodgement can also cause emboli that obstruct blood vessels in other parts of the body such as the brain, decreasing blood flow and oxygen supply to brain tissue and resulting in infarction.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
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    PATHOPHYSIOLOGY

    Non-modifiable Risk
    -Heredity
    -Race
    -Increasing age- 40 years & above
    -Gender- menopausal

    Modifiable Risk
    -smoking
    -hypertension
    -increase serum cholesterol level
    -physical inactivity

    Increase in the deposition of fat on


    blood vessel wall

    atherosclerosis

    Coronary artery

    CORONARY ARTERY
    DISEASE

    Decrease blood flow to the


    heart

    Stenosis of the arterial wall

    blood swirls around the


    irregular surface of the plaque

    Platelet adheres to the plaque

    Thrombus formation
    c/c: increase blood pressure
    Increase pressure on the walls
    of blood vessels
    HYPERTENSIVE
    CORONARY VASCULAR
    DISEASE

    Diagnostic test:
    -ECG-changes in ST segment
    and T wave
    -Coronary angiography
    -CT scan
    -MRI

    Increase peripheral vascular


    resistance
    Dislodge of plaque (emboli)

    Passes into the circulation


    going to other blood vessels
    especially on the brain

    Obstruction of blood vessels


    Decrease blood flow on the
    affected area

    Decreased O2 supply and


    nutrients

    BRAIN TISSUE
    INFARCTION
    C/C: extremity spasm

    Tissue injury

    triggers inflammatory response

    Increase injury trigger


    inflammatory response

    C/C: -episodes of vomiting


    -dizziness

    Increase ICP

    Diagnostic test:
    -CT scan
    -MRI

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