Referred Pain
Referred Pain
Referred Pain
Convergent-projection
believed that the internal organs were insensitive to stimuli. Furthermore, non-nociceptive
afferent inputs to the spinal cord created what termed "an irritable focus". This focus
caused some stimuli to be perceived as referred pain. An example of that ; are nerves
from damaged heart tissue which convey pain signals to spinal cord levels T1-T4
on the left side , which happen to be the same levels that receive sensation from
the left side of the chest and part of the left arm. The brain isnt used to receive
such strong signals from the heart , so it interprets them as pain in the chest &
left arm.
Axon-reflex
Axon reflex suggests that the afferent fiber is bifurcated before connecting to the dorsal horn.
Bifurcated fibers do exist in muscle, skin, and intervertebral discs. Yet these particular
neurons are rare and are not representative of the whole body. But Axon-Reflex also does not explain the
time delay before the appearance of referred pain, threshold differences for stimulating local and referred pain,
and somatosensory sensibility changes in the area of referred pain.
Hyperexcitability
HyperexcitabilityStimuli is transmitted to the corresponding ganglia Nerves which share the same ganglion
are stimulated Pain Stimuli is transmitted to these nerve fibers Pain is felt on fields supplied by those
nerves.
No summation takes place
Thalamic-convergence
Thalamic convergence suggests that referred pain is perceived as such due to the summation of neural inputs in
the brain, as opposed to the spinal cord, from the injured area and the referred area.
nearly the same mechanism as Hyperexcitability but with summation of peripheral stimuli.
Source of pain
Myocardial ischaemia
Left shoulder
Back
Pancreas
in which the vagus nerve or the trigeminal nerve in the throat and
the palate, respectively, transmit pain signals
Liver, gallbladder