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Community Acquired Pneumonia Pathophysiology

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VIII.

Pathophysiology

Pathophysiology of Community Acquired Pneumonia

Airborne droplets -

Precipitating Factor

Organisms (bacterial, viral)


streptococcus pneumonia
that contracted by
breathing

1.

Exposure to
chemical and
bacterial agents.

2.

Hospitalizations
because of
malaria.

Invasion of infection to
Enters nasal passage,
the Blood vessels in the
pharynx, larynx and
lung causing WBC and
trachea.
other defenses to come
as a result blood vessel
becomes
inflamed
host defense
wasand
cause
leakage
on
it.
Diminished or altered

Predisposing Factor
1. 50 years old
2. Filipino
3. history of
smoking, and
drinking alcohol
4. nature of work

by underlying diseases
and to unknown cause.

Causing these blood, debris, wbc and


other defences accumulated to the
alveoli and fill the normally aircontaining spaces.

Itchy sensation
on the throat

Infectious particle lies on


oropharynx w/c inflamed
and further result to
weakness or altered
This results in less
host defense
area for exchange of
oxygen and carbon
dioxide.

tend to become
solid because of
the debris and fluid
collection

This may also be


bloodborne organisms that
enter the pulmonary
circulation and are trapped
in the pulmonary
capillary
Increase
of mucus
bed, becoming
a
potential
production and
source of pneumonia.
mucus is sometimes
tinged the blood

Failure of the upper respiratoryObstruction on the


Patient is now deprived
of oxygen tract to prevent infectious
airway.
particle to reach the sterile
lower part of the lungs

of breathing,
Lungs Difficulty
become inflamed.
tachypnea, coughing
reflex.

Further leads to general


weakness
of dec.
causes the protein
rich fluidbecause
to seep into
alveoli.
WBC fighting the infection
also
fill the alveoli.
tissue
perfusion

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