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    COPD Patho

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    Glenn_Ancheta_2074
    COPD is caused by inhalation of pathogens like cigarette smoke that trigger an inflammatory response in the lungs. This leads to narrowing of the small airways due to injury and scar tissue formation. Over time, airflow becomes obstructed which causes shortness of breath, coughing, and other symptoms as oxygen levels decrease and carbon dioxide increases in the bloodstream.

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    COPD Patho

    Uploaded by

    Glenn_Ancheta_2074
    3K views1 page
    COPD is caused by inhalation of pathogens like cigarette smoke that trigger an inflammatory response in the lungs. This leads to narrowing of the small airways due to injury and scar tissue formation. Over time, airflow becomes obstructed which causes shortness of breath, coughing, and other symptoms as oxygen levels decrease and carbon dioxide increases in the bloodstream.

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    COPD patho

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    COPD is caused by inhalation of pathogens like cigarette smoke that trigger an inflammatory response in the lungs. This leads to narrowing of the small airways due to injury and scar tissue formation. Over time, airflow becomes obstructed which causes shortness of breath, coughing, and other symptoms as oxygen levels decrease and carbon dioxide increases in the bloodstream.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Download as doc, pdf, or txt
    3K views1 page

    COPD Patho

    Uploaded by

    Glenn_Ancheta_2074
    COPD is caused by inhalation of pathogens like cigarette smoke that trigger an inflammatory response in the lungs. This leads to narrowing of the small airways due to injury and scar tissue formation. Over time, airflow becomes obstructed which causes shortness of breath, coughing, and other symptoms as oxygen levels decrease and carbon dioxide increases in the bloodstream.

    Copyright:

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    Download as DOC, PDF, TXT or read online from Scribd
    Download as doc, pdf, or txt
    of 1

    PATHOPHYSIOLOGY OF COPD

    Etiology Predisposing Factors


    Streptococcus Pneumonae  smoking (110 pack
    years)
     age
     exposure to asbestos
    fiber, cotton fibers or

    Inhalation of pathogens

    Pharynx, nose, larynx and


    trachea
    (portal of entry)

    Inflammatory process

    Excess H2O and Injury RBC, fibrin and


    plasma protein polymorphonuclear leukocytes
    (Lower lobe) infiltrate the alveoli
    Narrowing of the small
    peripheral airways
    Irritates mucus Consolidation of leukocytes and
    membrane fibrin
    Injury-repair
    process Decreased Oxygen

    Accumulation of Scar tissue


    secretions Decreased tissue perfusion
    formation
    Narrowing of the
    Pale Hypoxemia Tachycardia
    airway lumen
    Airway Coughing/ DOB Hypoxia
    obstruction
    Inflammatory
    exudates Nasal Tachypnea
    Ventilation-
    progress flaring
    perfusion
    inequality
    Hyperemia
    Hypere
    Engorgement of the alveoli

    Space with fluids and Pulmonary edema


    hemorrhage exudates (Complication)

    Chest pain

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