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    Acute Anterior Wall Myocardial Infarction

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    Melinda Cariño Ballon
    An anterior wall myocardial infarction occurs when the anterior myocardial tissue supplied by the left anterior descending coronary artery suffers injury due to a lack of blood supply, usually from an occlusion in that artery. The patient experienced chest pain, tachycardia, shortness of breath, and anxiety. Treatment included oxygen, medication therapy, monitoring, and angioplasty and stent placement. Rehabilitation involved phases starting with limited activity in the hospital, progressing to increased activity at home with a rehabilitation team, and ultimately a long-term maintenance program to adopt healthy lifestyle habits.

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    Acute Anterior Wall Myocardial Infarction

    Uploaded by

    Melinda Cariño Ballon
    34 views2 pages
    An anterior wall myocardial infarction occurs when the anterior myocardial tissue supplied by the left anterior descending coronary artery suffers injury due to a lack of blood supply, usually from an occlusion in that artery. The patient experienced chest pain, tachycardia, shortness of breath, and anxiety. Treatment included oxygen, medication therapy, monitoring, and angioplasty and stent placement. Rehabilitation involved phases starting with limited activity in the hospital, progressing to increased activity at home with a rehabilitation team, and ultimately a long-term maintenance program to adopt healthy lifestyle habits.

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    Acute Anterior Wall Myocardial Infarction-

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    An anterior wall myocardial infarction occurs when the anterior myocardial tissue supplied by the left anterior descending coronary artery suffers injury due to a lack of blood supply, usually from an occlusion in that artery. The patient experienced chest pain, tachycardia, shortness of breath, and anxiety. Treatment included oxygen, medication therapy, monitoring, and angioplasty and stent placement. Rehabilitation involved phases starting with limited activity in the hospital, progressing to increased activity at home with a rehabilitation team, and ultimately a long-term maintenance program to adopt healthy lifestyle habits.

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    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
    34 views2 pages

    Acute Anterior Wall Myocardial Infarction

    Uploaded by

    Melinda Cariño Ballon
    An anterior wall myocardial infarction occurs when the anterior myocardial tissue supplied by the left anterior descending coronary artery suffers injury due to a lack of blood supply, usually from an occlusion in that artery. The patient experienced chest pain, tachycardia, shortness of breath, and anxiety. Treatment included oxygen, medication therapy, monitoring, and angioplasty and stent placement. Rehabilitation involved phases starting with limited activity in the hospital, progressing to increased activity at home with a rehabilitation team, and ultimately a long-term maintenance program to adopt healthy lifestyle habits.

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    ACUTE

    ANTERIOR
    MeaninWALL
    -An anterior wall myocardial infarction
    NURSING DIAGNOSIS
    g MYOCARD
    occurs
    usually
    when anterior myocardial tissue
    supplied by the left anterior -Acute Pain related to myocardial ischemia
    descending coronary artery suffers injuryresulting
    due from coronary artery occlusion.
    to lack of blood supply. -Ineffective Tissue Perfusion related to thrombus
    in coronary artery.
    IAL -Anxiety and Fear related to hospital admission
    and fear of death.

    CauseINFARCTIO Nursing Management


     Vasospasm. This is the sudden
    Placed on a semi-fowler’s position with Oxygen inhalation at 3-4
    constriction or narrowing of the


    N
    coronary artery.
    Decreased oxygen supply. 
    liters/ min via nasal cannula, Administer oxygen along with
    medication therapy to assist with relief of symptoms
    Assess pain status frequently with pain scale
     Increased demand for oxygen. 
    Assess hemodynamic status including BP, HR, LOC, skin color, and
    temperature (every 5 minutes during with pain; every 15 minutes)
    Clinical Monitor continuous ECG to detect dysrhytmias
    Perform 12-lead ECG immediately with new pain or changes in level
    -Chest pain -he describes it as “pain under
    my left chest that radiates to my left arm”. He rates of pain • Monitor respirations, breath sounds, and input and output to
    the pain as an 8 on a scale of 1 to 10. He looks detect early signs of heart failure
    diaphoretic and pale. Monitor O2 saturation and administer O2 as prescribed
    -Tachycardia and tachypnea. Keep client limited fluid intake at 800 cc/shift, on strict bed rest with
    -Sligth shortness of breath and crackles at
    the bases with. Because of increased
    oxygen demand and a decrease in the
    Medical
    no bathroom privileges with restrictions on having visitors.
    Provide a calm environment and reassure client and family to
    decrease stress, fear and anxiety
    PHASES OF
    supply of oxygen, shortness of breath occurs.

    REHABILITATI
    /Surgica
    Laboratory ON cardiogenic pulmonary l
    Pharmacologic Therapy- Daily medications are Aspirin 80 mg once
    daily after breakfast, Isosorbide dinitrate 10 mg 3x a day,
    Chest X ray shows
    Simvastatin 20 mg at bedtime, and Enalapril 10mg daily.
    edema.
    Findings
    FOLLOWING Phase I: Hospital
    12 lead ECG with progressive ST elevation in
    lead V2 to V5.
    MYOCARDIAL
    Troponin I Test result of 0.9 ng/mL,
    Interven Angioplasty and stent placement (percutaneous coronary
    revascularization)
    Coronary artery by pass surgery.

    INFARCTION tions
     Occurs while the patient is still hospitalised
     Activity level depends on severity of angina or MI
     Patient may initially sit up on bed or chair; perform range of motion exercise and self-care (walking, shaving) and
    progress to ambulation in hallway and limited stair climbing.
     Mr. MS was relieved from his anxiety after explaining his condition and treatment.

    Phase II: Early recovery

     Begins after the patient is discharged.


     Activity level is gradually increased and the supervision of the cardiac rehabilitation team and with ECG monitoring. He
    was monitored for signs of reperfusion: return of ST segment to baseline and reperfusion dysrhythmias
     Team may suggest that physical activity (e.g. walking) be initiated at home.
     Information regarding risk factor reduction is provided at this time.

    Phase III: Late recovery

     Long term maintenance program


     Individual physical activity programs are designed and implemented at home, a local gym or the rehabilitation centre.
     Patient and family possibly restructure lifestyle and roles.
     Lifestyle changes should become lifelong habits.
     Medical supervision is still recommended.

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