Symptomatic Apical Periodontitis Et Ology: Diagnosis and Treatment
Symptomatic Apical Periodontitis Et Ology: Diagnosis and Treatment
Symptomatic Apical Periodontitis Et Ology: Diagnosis and Treatment
PERIAPICAL TISSUES
m Cementum
m Periodontal ligament (PDL)
m Alveolar bone (Alveolar process)
PERIODONTIUM
§ Periodontium: The tissues that surround and support teeth, attaching them to the alveolar bone; includes bone, connective tissue,
vascular and neuronal elements.
§ Gingiva, Cementum, Periodontal ligament (PDL), Alveolar bone
INFLAMMATION / INFECTION
§ Inflammation: The cellular and vascular response of tissues to injury.
§ Acute inflammation: A response that is abrupt in onset and short in duration, characterized by the exudation of fluid, serum proteins,
inflammatory m diators and cells, mainly PMN into the area of injury. may become chronic if the injurious agent persists.
§ Chronic inflammation: A response that is slow in onset and of long-standing duration, characterized by proliferation of fibroblasts and
vascular endothelium and an influx of lymphocytes, plasma cells, macrophages and inflammatory mediators. It may be primary or
preceded by acute inflammatory response.
§ Infection: Invasion and proliferation of pathogenic microorganisms in body tissues and the reaction of the tissues to their presence.
APICAL INFLAMMATION / APICAL INFECTION
§ Apical inflammation periapical tissue reaction to irritants emerging from the root canal system that manifests as vasodilation,
increased vascular permeability, and exudation.
§ In contrast apical infection is due to the physic presence of pathogenic microorganisms in the periapical tissues that subsequently
produce tissue damage.
§ In diseases caused by infection, bacteria are usually present in the involved tissues or organs.
§ Although apical periodontitis is primarily an infectious disease, bacteria are usually not present in the periapical tissues but in the root
canal system (except for extraradicular endodontic infection).
Pathological changes in the periapical region are defined as "periradicular / periapical diseases".
à Occlusal Trauma with thickening of the lamina dura, widening of the periodontal space and increased diffuse periodontal bone density
(red arrow), with vertical bone loss (green arrows)
§ Acute trauma
o Mostly extrusion and lateral luxation cases
§ Extension of irreversible pulpitis in the late stage into the PDL / periapical tissues through the apical foramen
TREATMENT
√ Root canal treatment
Iatrogenic problems during root canal treatment
MECHANICAL, CHEMICAL AND PHYSICAL IRRITATION OF THE PERIAPICAL TISSUES
§ Inaccurate determination of the root canal working length
§ Disruption/widening of the apical construction during instrumentation
§ Exiting the periapical tissues with canal instruments (over instrumentation)
§ During the root canal treatment of irreversible pulpitis
§ Exiting the periapical tissues with canal instruments (over instrumentation); since the patient is under anesthesia, he/she does not
feel pain and does not react. The bleeding continues.
§ Because the periapical tissues are intact, when the root canal instrument comes out of the apex; mechanical irritation immediately
causes symptomatic apical periodontitis.
§ Patient's complaint: I have a lot of pain, I feel like my tooth has elevated, when I close my mouth, it feels like that tooth touches the
first, I can't even speak...
EXTRUSION OF MICROORGANISM OR TOXINS INTO THE PERIAPICAL TISSUES
§ Disruption/widening of the apical construction during instrumentation
§ Exiting the periapical tissues with canal instruments (over instrumentation)
§ Overextension of the microorganisms or their toxins within the root canal into the intact periapical tissues causes symptomatic apical
periodontitis.
MECHANICAL, CHEMICAL AND PHYSICAL IRRITATION OF THE PERIAPICAL TISSUES
§ In cases with intact periapical tissues;
o Overextension of root canal filling materials, sealer and/or gutta-percha cause chemical and phycical damage.
o Extrusion of root canal irrigation solutions (eg. NaOCl) or intracanal medicaments (eg. Ca(OH)2) cause chemical damage and
symptomatic apical periodontitis develops.
(a) Preoperative periapical radiograph, (b) clinical view after curettage of periapical lesion (c) clinical view showing root
capping with mineral trioxide aggregate (MTA) (Ins shows the resected overfilled g a-percha portion) d) Immediate post-
operative periapical radiograph following MTA retrograde filling