Location via proxy:   [ UP ]  
[Report a bug]   [Manage cookies]                

Unit 3 Tutorial Questions 1. Gestational Diabetes

Download as docx, pdf, or txt
Download as docx, pdf, or txt
You are on page 1of 2

Unit 3 Tutorial questions

1. Gestational diabetes
a. Rationalize the three classic signs of gestational diabetes.
 Polydipsia (excessive thirst): it is caused by increase glucose level. When blood glucose
level get high the kidney produce urine in an effort to remove extra glucose from the
body. Meanwhile, because the body loos fluids we tend to drink in order to replace
them. This lead to the feeling of intense thirst associated with diabetes.

 Polyphagia (excessive hunger): in people with diabetes glucose can’t enter the cell to be
used for the energy. This is due to the low insulin level or insulin resistance. This is
because the body can’t convert this glucose into energy you will begin to feel very
hungry. Hunger associated with this does not go away unless we consume food.

 Polyuria (frequent urination): when the glucose level are too high, pur body will try to
remove some of the excess glucose via urination. This lead to our kidney filtering out
more water which lead to increase need to urinate.

b. Rationalize macrosomic babies of gestational diabetic mothers.


This is due to the high insulin resistance of the mother. A higher amount of blood glucose passes
through the placenta into the fetal circulation. As a result, extra glucose in the fetus is stored as body fat
causing macrosomia, which is also called 'large for gestational age'.

c. Explain the management and the complication of diabetes on;


maternal and foetus.
Women with diabetes do not have the capacity to increase insulin secretion in response to the
altered carbohydrate metabolism in pregnancy and therefore glucose accumulates in the
maternal and fetal system leading to significant morbidity and mortality.

Close monitoring of the mother blood glucose is done to reduce the risk of long-term
complication. Blood glucose can also be estimated by testing urine for glucose using reagent
straps, although this is less accurate than the blood test.
2. Gestational hypertension/Pregnancy induced hypertension
a. Describe the following types of hypertensions;
severe hypertension and gestational hypertension.
Gestational hypertension this is a form of high blood pressure in pregnancy and it exist only
during pregnancy period. The blood pressure should return to normal value postnatally. Severe
hypertension this can be described as when the systolic blood pressure level is 160mmHg or in
other words higher and the diastolic pressure is 110mmHg or greater
b. Discuss the management of pre-eclampsia
Thorough investigations of underlying medical problems. Early recognition of pre-eclampsia
help also in it’s early treatment. Hospital admission and referral and therefore treatment of
hypertensions is done. Expedite the birth of baby and placenta. Induction of labor at different
gestational age blood pressure level. Corticosteroids (side effects ) and liaison with neonatal
intensive care and anesthetic team.

c. Discuss the management of severe eclampsia


Mother is put on rest as much as needed. Ensure that diet given to the mother are rich in
protein, fiber and vitamins. Weight gain daily check up is done and also blood pressure is
checked and urinalysis. Abdominal examination is done on daily basis. Foetal assessment 8s
monitored through kick chart, CTG monitoring, ultrasound scan. Dappled flow studies to
determine the placental blood flow.

3.Cardiac diseases in Pregnancy


a. Explain the functional classification of the cardiac diseases.
b. Discuss the management of cardiac mothers under prenatal care, intrapartum care
and postnatal care
4. Other conditions – Anemia, STI, Renal, Epilepsy

a. Explain the causes and management of Folic Acid anemia in pregnancy.


b. Discuss the cause, sign and symptom and management of syphilis.
c. Explain the effects renal disorders in pregnancy.
d. Discuss the clinical management epilepsy in pregnancy.

REFERENCE
Medically reviewed by Marina Basina, M.D. — Written by Jill Seladi-Schulman, Ph.D. — Updated on
June 18, 2020

You might also like