Eating Disorder 1
Eating Disorder 1
Eating Disorder 1
Roles of
neurotransmitters
Strong genetic
influences
Social stress
THE EXACT CAUSE OF
ANOREXIA IS UNKNOWN
PSYCHOL ENVIRON
OGICAL MENTAL
GENES CULTURAL
DIAGNOSTICS
Inappropriate exercise.
GASTROINTESTINAL
COMPLICATIONS
HORMONAL CHANGES
RELATED TO THE
HYPOTHALAMIC-PITUITARY-
GONADAL AXIS
BRAIN ATROPHY
PHYSICAL AND
LABORATORY
ASSESSMENT OF
EATING DISORDERS
MANAGEMENT
What are the desired
outcomes?
Patients receiving
A reduction in the
SSRIs should be
frequency and severity
monitored.
of abnormal eating
habits,
A diary especially in the
outpatient setting.
monitoring
Follow-up laboratory tests and ECGs are not part of routine
monitoring unless the patient is restricting food intake, is
purging, or continues to lose weight despite treatment. A
healthy weight gain of no more than 0.2 to 0.5 kg (0.4 to 1.1 lb)
per week toward a goal of 90% to 95% of normal weight or a
BMI greater than 18.5 kg/m 2 is a critical sign of treatment
success. A patient’s use of coping skills and contingencies for
dealing with stress other than manipulating food consumption
also should be assessed.
CASE STUDY
Emma’s parents were worried about their 16 year old daughter; for
over a year she had been on a diet that didn’t seem to stop. Emma's
dieting behaviours made her more withdrawn, depressed and
anxious. one day, her parents received a phone call from school to
say that Emma had fainted and was in hospital. She was
immediately admitted to a hospital as an inpatient because her
weight was dangerously low. Emma also had a very low heart rate
(bradycardia) and a low temperature (hypothermia).
FINDINGS ASSESSMENT
Underweight A thorough assessment of
PHARMACOLOGICAL
Antidepressants
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