Peptic

ulcer mcqs pdf

What is peptic ulcer how is peptic ulcer caused class 10. Peptic ulcer menu. Types of peptic ulcer pdf.

Q1. Which pathogen is strongly associated with peptic ulcer? (a) Bifidobacterium bifidum (b) Escherichia coli (c) Helicobacter pylori (d) Faecalibacterium prausnitzii Q2. Gastric ulcers are most common in younger patients (a) True (b) False Q3. How do NSAIDs contribute to peptic ulcer formation?
(a) By burning holes directly in the gastric mucosa when they hit the stomach. (b)Increasing acid production (c) Decreasing prostaglandin synthesis HOME HEALTH TOPICS Healthy Living SYMPTOMS EMERGENCIES RESOURCES NEWS ABOUT 10 Questions | Attempts: 61514 Which of the following is NOT a causative agent for a peptic ulcer?
What are the complications of a Peptic Ulcer?
When HP is the cause of PUD, the ulcer disease can be eradicated with The lifetime risk of ulcer disease is Both gastric ulcers and H.pylori infection are highly associated with _________. Ulcerations typically occur in regions bathed with acid/pepsin, such as the What often describes the pain of PUD? Sharp, knife-like. Radiates to the right shoulder
Dull, achy. Localized to the stomach. Diffuse, joint arthralgia. Burning or gnawing. Radiates to the back. Complications of PUD include bleeding, perforation, and penetration.
1.) Bleeding typically manifests as _________________. 2.) PUD is the most common cause of nonhemorrhagic GI bleeds/ What is best used for detecting small or healing ulcers? What is the treatment for PUD in addition to discontinuing irritating factors? Bismuth subsalicylate plus tetracycline Stomach Bacteria Influenza Abdomen In order to continue
enjoying our site, we ask that you confirm your identity as a human. Thank you very much for your cooperation. This is a quiz that contains NCLEX review questions for peptic ulcer disease. As a nurse providing care to a patient with peptic ulcer disease, it is important to know the signs and symptoms, pathophysiology, medications, nursing
management, diet education, and complications. In the previous NCLEX review series, I explained about other GI disorders you may be asked about on the NCLEX exam, so be sure to check out those reviews and quizzes as well. (NOTE: When you hit submit, it will refresh this same page. Scroll down to see your results.) Peptic Ulcer Disease NCLEX
Questions 1. In the stomach lining, the parietal cells release _________ and the chief cells release __________ which both play a role in peptic ulcer disease. A. pepsin, hydrochloric acid B. pepsinogen, pepsin C. pepsinogen, gastric acid D. hydrochloric acid, and pepsinogen 2. A patient has developed a duodenal ulcer. As the nurse, you know that which of
the following plays a role in peptic ulcer formation. Select ALL that apply: A. Spicy foods B. Helicobacter pylori C. NSAIDs D. Milk E. Zollinger-Ellison Syndrome 3. You’re educating a group of patients at an outpatient clinic about peptic ulcer formation. Which statement is correct about how peptic ulcers form? A. “An increase in gastric acid is the
sole cause of peptic ulcer formation.” B.

“Peptic ulcers can form when acid penetrates unprotected stomach mucosa. This causes histamine to be released which signals to the parietal cells to release more hydrochloric acid which erodes the stomach lining further.” C. “Peptic ulcers form when acid penetrates unprotected stomach mucosa. This causes pepsin to be released which signals to
the parietal cells to release more pepsinogen which erodes the stomach lining further.” D. “The release of prostaglandins cause the stomach lining to breakdown which allows ulcers to form.” 4. Your patient is diagnosed with peptic ulcer disease due to h.pylori. This bacterium has a unique shape which allows it to penetrate the stomach mucosa. You
know this bacterium is: A. Rod shaped B. Spherical shaped C. Spiral shaped D. Filamentous shaped 5. Helicobacter pylori can live in the stomach’s acidic conditions because it secretes ___________ which neutralizes the acid. A. ammonia B. urease C.
carbon dioxide D. bicarbonate 6. The physician orders a patient with a duodenal ulcer to take a UREA breath test. Which lab value will the test measure to determine if h.
pylori is present? A. Ammonia B. Urea C.
Hydrochloric acid D. Carbon dioxide 7. A patient arrives to the clinic for evaluation of epigastric pain. The patient describes the pain to be relieved by food intake. In addition, the patient reports awaking in the middle of the night with a gnawing pain in the stomach. Based on the patient’s description this appears to be what type of peptic ulcer? A.
Duodenal B. Gastric C.
Esophageal D. Refractory 8. A patient with chronic peptic ulcer disease underwent a gastric resection 1 month ago and is reporting nausea, bloating, and diarrhea 30 minutes after eating.
What condition is this patient most likely experiencing? A. Gastroparesis B. Fascia dehiscence C. Dumping Syndrome D. Somogyi effect 9. Thinking back to the patient in question 8, select ALL the correct statements on how to educate this patient about decreasing their symptoms: A. “It is best to eat 3 large meals a day rather than small frequent
meals.” B. “After eating a meal lie down for 30 minutes.” C. “Eat a diet high in protein, fiber, and low in carbs.” D. “Be sure to drink at least 16 oz. of milk with meals.” 10. A patient is recovering from discomfort from a peptic ulcer. The doctor has ordered to advance the patient’s diet to solid foods. The patient’s lunch tray arrives. Which food should
the patient avoid eating? A. Orange B. Milk C. White rice D. Banana 11. Which statement is INCORRECT about Histamine-receptor blockers? A.

“H2 blockers block histamine which causes the chief cells to decrease the secretion of hydrochloric acid.” B. “Ranitidine and Famotidine are two types of histamine-receptor blocker medications.” C. “Antacids and H2 blockers should not be given together.” D. All the statements are CORRECT. 12. You are providing discharge teaching to a patient
taking Sucralfate (Carafate). Which statement by the patient demonstrates they understand how to take this medication? A. “I will take this medication at the same time I take Ranitidine.” B. “I will always take this medication on an empty stomach.” C. “It is best to take this medication with antacids.” D. “I will take this medication once a week.” 13.
Select all the medications a physician may order to treat a H. Pylori infection that is causing a peptic ulcer? A. Proton-Pump Inhibitors B. Antacids C. Anticholinergics D. 5-Aminosalicylates E. Antibiotics F. H2 Blockers G. Bismuth Subsalicylates 14.

A physician prescribes a Proton-Pump Inhibitor to a patient with a gastric ulcer. Which medication is considered a PPI? A. Pantoprazole B. Famotidine C. Magnesium Hydroxide D. Metronidazole 15. A patient with a peptic ulcer is suddenly vomiting dark coffee ground emesis. On assessment of the abdomen you find bloating and an epigastric mass in
the abdomen. Which complication may this patient be experiencing? A. Obstruction of pylorus B. Upper gastrointestinal bleeding C. Perforation D. Peritonitis Answer Key: 1. D 2. B, C, E 3. B 4. C 5. B 6.

D 7. A 8. C 9. B, C 10. A 11. A 12.

B 13. A, E, F, G 14. A 15.

B You're Reading a Free Preview Page 2 is not shown in this preview. Drug Biotechnology Multiple Choice Questions & Answers on “Pharmacotherapy of Peptic Ulcer – 2”. 1. Which enzyme of H. pyloric helps it to grow in the acidic medium of the stomach?a) Ureaseb) Amylasec) Proteased) Acid HydrolasesAnswer: aClarification: H. pylori primarily
colonize in the antrum of the stomach it resides mainly within the gastric mucus. H. pylori have a high activity of the enzyme urease which enables it to colonize in the stomach. Infection can spread through fecal route. Infection thought to occur between parents and young children. 2. How to diagnose infection by H. pylori?a) Histology and cultureb)
Antibody testc) Urease CLO testd) Antibody test, Urease CLO test, Histology and cultureAnswer: dClarification: The majority of cases of peptic ulcer disease are related to H. pylori. The diagnosis of H. pylori infection must be confirmed prior to initiation of therapy. The methods can be histology and culture, antibody test, urease CLO test. H. pylori
have a high activity of the enzyme urease which enables it to colonize in the stomach. Infection can spread through fecal route.
Infection thought to occur between parents and young children. 3. Which cells are also known as chief cells?a) Parietal cellsb) Mucous neck cellsc) Peptic cellsd) Superficial epithelial cellsAnswer: cClarification: Peptic cells in the stomach lining is also known as a chief cell. The stomach lining has parietal cells that secrete acid, Goblet cells, mucous
neck cells, superficial epithelial cells.
All these cells can be found in the body and fundus of the stomach. 4. How neuronal regulation of gastric acid is done?a) Acetylcholineb) Gastrinc) Histamined) CNSAnswer: aClarification: Neuronal control is done by secretion of acetylcholine by the cholinergic neuron. Hormonal control of gastric acid is done by gastrin. Paracrine control of gastric
acid is done by histamine secreted by enterochromaffin-like cells. 5.
How hormonal regulation of gastric acid is done?a) Acetylcholineb) Gastrinc) Histamined) CNSAnswer: bClarification: Neuronal control is done by secretion of acetylcholine by the cholinergic neuron. Hormonal control of gastric acid is done by gastrin. Paracrine control of gastric acid is done by histamine secreted by enterochromaffin-like cells. 6.
Which will be the correct option for the marked place?a) Acetylcholineb) Gastrinc) Histamined) Hydrogen ionsAnswer: aClarification: Cholinergic neuron secretes acetylcholine that through circulation goes and bind to the muscarinic receptor in the parietal cell secrete acid into the gastric lumen.
Enterochromaffin-like cells secrete histamine which again goes and binds to the H2 receptor and through the cAMP pathway helps the parietal cell to secrete acid. Gastrin secreted by Antral G cell is the hormonal control of acid secretion by the parietal cell.
Gastrin, Histamine, and acetylcholine switch on the proton pump thus pumping H+ ions to produce acid in the gastric lumen.
7. Which will be the correct option for the marked place?a) Mucous cellsb) Parietal cellsc) Enterochromaffine like cellsd) Antral G cellAnswer: bClarification: Cholinergic neuron secretes acetylcholine that through circulation goes and bind to the muscarinic receptor in the parietal cell secrete acid into the gastric lumen.
Enterochromaffine like cells secrete histamine which again go and bind to the H2 receptor and through the cAMP pathway helps the parietal cell to secrete acid. 8. Which will be the correct option for the marked place?a) Acetylcholineb) Gastrinc) Histamined) AcidAnswer: dClarification: Enterochromaffine like cells secrete histamine which again go
and bind to the H2 receptor and through the cAMP pathway helps the parietal cell to secrete acid. Gastrin secreted by Antral G cell is the hormonal control of acid secretion by parietal cell. Acid in our stomach is secreted by the parietal cells. Mucous is secreted by the goblet cells. 9. Which will be the correct option for the marked place?a)
Acetylcholineb) Gastrinc) Histamined) Hydrogen ionsAnswer: bClarification: Gastrin secreted by Antral G cell is the hormonal control of acid secretion by parietal cell. Gastrin, Histamine and acetylcholine switch on the proton pump thus pumping H+ ions to produce acid in the gastric lumen. Acid in our stomach is secreted by the parietal cells.
Mucous is secreted by the goblet cells. 10. Which will be the correct option for the marked place?a) Mucous cellsb) Red blood cellc) Enterochromaffine like cellsd) Antral G cellAnswer: cClarification: Enterochromaffine like cells secrete histamine which through circulation again go and bind to the H2 receptor and through the cAMP pathway helps the
parietal cell to secrete acid.
Gastrin secreted by Antral G cell is the hormonal control of acid secretion by parietal cell. 11. Which will be the correct option for the marked place?a) Acetylcholineb) Gastrinc) Histamined) Hydrogen ionsAnswer: cClarification: Cholinergic neuron secretes acetylcholine that bind to the muscarinic receptor in the parietal cell secrete acid into the
gastric lumen. Enterochromaffine like cells secrete histamine which again bind to the H2 receptor and through the cAMP pathway helps the parietal cell to secrete acid. Gastrin secreted by Antral G cell is the hormonal control of acid secretion by parietal cell. 12. Which will be the correct option for the marked place?a) Mucous cellsb) Red blood cellc)
Enterochromaffine like cellsd) Antral G cellAnswer: dClarification: Cholinergic neuron secretes acetylcholine that through circulation goes and bind to the muscarinic receptor in the parietal cell secrete acid into the gastric lumen. Enterochromaffine like cells secrete histamine which again go and bind to the H2 receptor and through the cAMP
pathway helps the parietal cell to secrete acid. Gastrin secreted by Antral G cell is the hormonal control of acid secretion by parietal cell. Gastrin, Histamine and acetylcholine switch on the proton pump thus pumping H+ ions to produce acid in the gastric lumen.
13. Inhibition of H+, K+-ATPase blocks both basal and stimulated acid secretion.a) Trueb) FalseAnswer: aClarification: H+, K+-ATPase is located in the apical membrane of the oxyntic cell along the secretory canaliculi. The pump requires large amounts of energy that is supplied by intracellular ATP that is why inhibition of H+, K+-ATPase blocks
both basal and stimulated acid secretion. 14. Cimetidine is an H1 receptor antagonist.a) Trueb) FalseAnswer: bClarification: Cimetidine is an H2 receptor antagonist thus used in the treatment of peptic ulcer. It inhibits basal acid secretion of the stomach including nocturnal secretion. It is readily absorbed after oral administration and has a brief
duration of action from 4-8 hr. To practice all areas of Drug Biotechnology,