Spinal Cord Injuries
Spinal Cord Injuries
Spinal Cord Injuries
Introduction :
Management of spinal cord injured patient requires
specialized knowledge beyond the scope of any single person ,
so the team effort is required including –(the primary managing
physician, physical therapist, occupational therapist , nurse),
social worker , psychologist , vocational counselors and trainers
This team should work throughout the patients rehabilitation ,
including outpatient follow –up .The excellent environment for
such management is the spinal cord injury center , where
patients can be treated from the day of injury to the ultimate
vocational rehabilitation.
Clinical picture:
1- Spinal shock:
Immediately following SCI, there is a period of areflexia. It
results from abrupt withdrawal of connections between
higher centers and the spinal cord. It is characterized by
absence of all reflex activity, flaccidity and loss of sensation
below the level of lesion.
2- Motor deficits and sensory loss:
Following spinal cord injury, there will be either complete or
partial loss of muscle function below the level of the lesion.
Disruption of the ascending sensory fibers following SCI
results in impaired or absent sensation below the lesion level.
The clinical presentation of motor and sensory deficits is
dependent on the specific features of the lesion. These
include the neurologic level, the completeness of the lesion,
the symmetry of the lesion (transverse or oblique) and the
presence or absence of sacral sparing.
4- Respiratory impairment:
5-Spasticity:
It typically occurs below the level of lesion after spinal
shock subsides. There is a gradual increase in spasticity during
the first six months and usually reaches a plateau one year after
injury.
6- Bladder dysfunction:
The spinal integrating center for micturition is the conus
medullaris The type of bladder function remaining depends on
the location of the lesion – whether it is at , above , or below the
conus medullaris.
Spastic _ upper motor neuron bladder _ reflexly empties
when bladder is full and occurs in injuries above T12 (similar to
an infant's bladder ) . The reflex arc is intact.
Flaccid _ lower motor neuron bladder ¬ emptying is
initiated by pressure on abdomen , straining , A flaccid bladder
occurs in complete L1 paraplegics (cauda equine injury)
7- Sexual dysfunction:
Function depends on the presence or absence of reflex activity
and is closely linked to the type of bladder the patient exhibits .
Spastic _ upper motor neuron bladder patient _ males may
have non psychic erections that are reflex in nature . they may
be spontaneous or set off by mechanical stimulation , they may
or may not ejaculate and father children. Females may conceive
and give birth.
Flaccid _ lower motor neuron bladder patient _ Males
usually do not have erections or ejaculations and , therefore ,
demonstrate less function than spastic patients .
*Secondary complications:
1. Pressure sores: Pressure sores are subjected to infection
which may migrate to bone. It is a major cause of delayed
rehabilitation and may lead to death. *The most important
factors in the development of pressure sores are:
a) Impaired sensory function
b) Inability to make appropriate positional changes
c) Loss of vasomotor control, which lowers tissue resistance to
pressure.
d) Spasticity, with resultant shearing forces between bony
surfaces.
e) Skin maceration from exposure to moisture (e.g. urine).
f) Nutritional deficiencies as low serum protein and anemia.
g) Secondary infections.
Areas susceptible to pressure in recumbent position.
5. Contractures:
Contractures develop with prolonged shortening of
structures across and around a joint, resulting in limitation in
motion. Contractures initially produce alterations in muscle
tissue but rapidly progress to involve capsular and pericapsular
changes.
6. Deep venous thrombosis (DVT): They may block pulmonary
vessels and result in death. In SCI, DVT is due to loss of the
normal pumping mechanism provided by active contraction of
lower extremity musculature. This slows the flow of blood,
allowing higher concentrations of pro-coagulants (thrombin) to
develop in localized areas.
Prognosis
IV – Ambulation:
b) Crutch balance
c- Crutch walking
C) Sitting up – holding on
D) Sitting up-holding on to two ropes
9) Crawling
Purpose: To strength arm, shoulder and thigh muscles.
To maintain balance while moving forward
Preparation For: Crutch walking.
Starting position: Quadribed.
10) Raising arms Alternately in a 4 legged position
Purpose: To maintain balance on knees and one hand.
Preparation For: Crutch balance, Crutch walking.
Starting positio : Quadribed position
II ) Wheel Chair:-
Description of wheel chair:
Wheel chair prescription will vary according to the level of
lesion.
• Seat depth: 2.5cm back from popliteal fossa.
• Floor –to- seat height: 2 inches clearance from the
floor to the foot pedals and 90o knee flexion.
• Back height: low back for paraplegia, high back that is
below the inferior angle of the scapula to provide push-
ups activities.
• Seat width: hand's width between the hips and the sides
of the chair.
Wheel chair activities :
Exercise's done on wheel chair:-
1- Sitting balance.
2- Push-ups.
3- Crossing legs.
4- From wheel chair to mat and back.
4) From wheel chair to mat
a) using 6 balances b) directly
Purpose: To strength, balance to able to get down on mat or
floor without being lefted.
Preparation For: Transfer from wheel chair to toilet seat and
back.
Starting position: Sitting on wheel chair , graduated 6 balances
IV – Ambulation: