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Atlas of Cosmetic and Reconstructive Periodontal
Surgery 3 E 3rd Edition Edward S. Cohen Digital Instant
Download
Author(s): Edward S. Cohen
ISBN(s): 9781550092677, 1550092677
Edition: 3
File Details: PDF, 76.03 MB
Year: 2006
Language: english
Cohen__i-xii_FM.qxd 11/23/06 4:20 PM Page i
ATLAS OF
COSMETIC AND
RECONSTRUCTIVE
PERIODONTAL
SURGERY
Cohen__i-xii_FM.qxd 11/23/06 4:20 PM Page ii
Cohen__i-xii_FM.qxd 11/23/06 4:20 PM Page iii
ATLAS OF
COSMETIC AND
RECONSTRUCTIVE
PERIODONTAL
SURGERY
THIRD EDITION
2007
BC Decker Inc EXIT
Hamilton
Cohen__i-xii_FM.qxd 11/23/06 4:20 PM Page iv
BC Decker Inc
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Notice: The authors and publisher have made every effort to ensure that the patient care recommended herein, including choice of drugs and drug dosages, is in accord
with the accepted standard and practice at the time of publication. However, since research and regulation constantly change clinical standards, the reader is urged to
check the product information sheet included in the package of each drug, which includes recommended doses, warnings, and contraindications. This is particularly
important with new or infrequently used drugs. Any treatment regimen, particularly one involving medication, involves inherent risk that must be weighed on a case-
by-case basis against the benefits anticipated. The reader is cautioned that the purpose of this book is to inform and enlighten; the information contained herein is not
intended as, and should not be employed as, a substitute for individual diagnosis and treatment.
Cohen__i-xii_FM.qxd 11/23/06 4:20 PM Page v
Dedicated to
Meyer and Milton
Cohen__i-xii_FM.qxd 11/23/06 4:20 PM Page vi
Contributors
Contents
Preface . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . ix
PART I Basics
SECTION 1 Fundamentals
1. Prognosis . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 1
2. Surgical Basics . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 9
3. Sutures and Suturing . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 15
4. Scaling, Root Planing, and Curettage . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 29
5. Gingivectomy . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 39
6. Mucogingival Surgery . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 45
7. Palatal Flaps . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 87
8. Cosmetic Treatment of Maxillary Anterior Teeth . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 103
viii Contents
Bibliography . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 439
Index . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 447
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Preface
Periodontics is both an art and a science, and this textbook is dedicated to the art of periodontics. The goal
of this atlas is to teach the novice, upgrade the skills of the average clinician, and act as a reference source
for the experienced clinician.
The modern paradigm for periodontal surgery has significantly changed since the last edition. Esthetics
and implantology are now the cornerstones of the modern periodontal practice.
Dental esthetics has altered the way we view our cases. No longer do we treat cases without consider-
ation being given to the facial, dentofacial, and dentogingival elements, especially in the esthetic zone. Pro-
cedures have been developed and refined to maintain, augment, and alter the dentogingival elements for
the purpose of achieving a satisfactory esthetic result.
Dental implants, although greatly expanding our treatment options, have significantly impacted neg-
atively upon the art of periodontics. Too often teeth are now prematurely extracted for implant placement.
As a consequence, clinical skills are reduced and the learning curve is expanded, further reinforcing extrac-
tion over treatment. This will change only when there is a greater emphasis placed on treatment and
preservation, which is the goal of this atlas.
No textbook of this kind can be completed without the help of others. In that regard, I must take spe-
cial note to thank Drs. Dennis Shanelec and Leonard S. Tibbits for their section on microsurgery, Drs.
Arun K. Garg and Craig Misch for their assistance with the section on mandibular chin and ramus grafts,
Dr. Eiji Funakoshi for his section on enamel matrix derivatives and his assistance with the section on
osteotomes, Dr. James Hanratty for his clinical contributions, especially to the chapters on mandibular
chin grafts and sinus lifts, and Dr. Periklis Proussaefs for his contribution of the Loma Linda technique. I
would also like to thank Drs. Scott Kissel, Roger Wise, Federico Brugnami, Irving Glickman, Kenneth
Kornman, and George Goumenous for their clinical contributions. Any omissions of recognition are acci-
dental and will be corrected in any future editions.
It must be noted that the chapters written on dental esthetics, esthetic diagnosis, and fundamentals of
esthetics relied in large part on the following source material: The Principles of Visual Perception and Their
Clinical Application to Denture Esthetics by Richard E. Lombardi, Esthetics of Anterior Fixed Prosthodontics
by Gerard J. Chiche and Alain Pinault, and Fundamentals of Esthetics by Claude R. Rufenacht.
I wish to thank the models for this edition of the atlas, Shanon O’Brien-Cohen, Christine Watson,
Judith Cohen, and Brigette Deveraux. Their help was greatly appreciated.
Lastly, special recognition must be given to Robert Ullrich, without whose artwork this and the pre-
vious atlases could not have been completed. He is a master medical illustrator whose work has been
copied in every textbook and atlas on periodontal surgery.
Edward S. Cohen
Boston, Massachusetts
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This surgical atlas was originally published with the intent of being the most complete periodontal surgi-
cal atlas and in 1988 it was. Since that time, there have been many important advances. The emphasis in
periodontics has clearly shifted toward reconstructive periodontics. Guided tissue regeneration, biome-
chanical root preparation, predictable bone regeneration procedures, and cosmetic root coverage have
made reconstructive periodontics a reality.
This edition will reflect these changes with new chapters on biomechanical root preparation, guided
tissue regeneration, cosmetic gingival reconstruction, cosmetic treatment of the maxillary anterior teeth, and
ridge augmentation, and expansion of the chapter on inductive osseous surgery. A new chapter has also been
added on sutures and suturing. All other chapters have been brought up to date, again with the intention
of again making this the most complete periodontal surgical atlas.
Any book of this kind requires the help of others in order to be completed. In this regard, a special
thanks must go to all those clinicians who so unselfishly contributed material for this edition (alphabeti-
cally): Burton Becker, Gerald M. Bowers, Daniel Buser, Robert Del Castillo, Stuart Froum, Bernard
Gantes, Gary Golovic, Jan Gottlow, Claude G. Ibbot, L. Laurell, James T. Mellovig, Sture Nyman, Knut
Selvig, Richard H. Shanaman, Athenos Spiros, Sigmond Stahl, Dennis P. Tarnow, and Theodore West.
Special acknowledgments must be extended to W.R. Gore Associates, Flagstaff, AZ, and Guidor AB,
Gothenburg, Sweden (Guided Tissue Regeneration, Chapter 13) and Ethicon, Inc. Somerville, NJ (Sutures
and Suturing, Chapter 2) for their help and permission for parts of their clinical manuals to be incorpo-
rated into this atlas.
To my dear friends and associates BobUllrich (artwork) and Harry Maskell (photography), without
whose talent and expertise this book most surely would not have been completed, I say again thank you.
Edward S. Cohen
Boston, Massachusetts
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Periodontology is both an art and science; as practiced daily, however, it is predominantly a surgical spe-
cialty. Although the major periodontal textbooks contain surgical sections, their general nature and scope
do not allow for an in-depth analysis of any one specific area. It is for this reason that this text is devoted
solely to the art of periodontics and designed for the student, general practitioner, and specialist.
Each procedure has been illustrated and laid out in a step-by-step fashion. Clinical examples have
been used secondarily only to supplement illustrations. The descriptive nature of the text is meant to be
both brief and simple. Each chapter presents indications, contraindications, advantages, disadvantages,
and related problems for each procedure.
This atlas incorporates most of the general techniques and concepts’ that are outlined in the major
textbooks. It can, therefore, easily be used as a supplement to any of these textbooks.
In the course of writing this text, careful attention has been paid to faithfully describing the proce-
dures as they were outlined originally, as well as attempting to give credit to their originators. Any over-
sights are unintentional and would gladly be corrected in the future. In this regard credit must be given
to Glickman’s Clinical Periodontology for serving as the model to base the drawings of gingivectomy on
and Lindhes: Clinical Periodontology for serving as a guide for the chapter on furcations.
I would like to thank my colleagues Edward Allen, Raul Caffesse, Jose Carvalho, Giovanni Castellucci,
David Garber, Barry Jaye, and Edwin Rosenberg for their clinical contributions; Mark Hirsh, Mayer Liebman,
and Peter Ferrigno for their helpful suggestions; and my assistants Jeanne McCormack, Rebecca Mugherini,
Christine Roberts, and Judith Cohen for their help.
Special notes of acknowledgment must be given to Harry W. Maskell for his photographic excellence;
to Robert H. Ullrich, Jr., medical illustrator, for his creative genius in the designing and drawing of the
illustrations; and to the educational media department of the New England Medical Center for the picto-
rial overlays.
Edward S. Cohen
Boston, Massachusetts
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Cohen_001-008_01.qxd 11/16/06 8:19 PM Page 1
Prognosis
The term prognosis has been used to indicate the Newman and colleagues (1994) stated that Table l-1 Periodontal Risk Factors
prediction of the future course of a disease in terms the terms risk and prognosis are interchangeable,
of disease outcomes following its onset and/or treat- with “risk most often thought of as the probabil- Smoking
ment. As clinicians, we are constantly asked to ity of getting the disease (initiation) or having Subgingival plaque
evaluate the short- and long-term prognosis of the disease get worse (disease progression).” Gingival color changes
Initial attachment loss
both the individual tooth and the overall denti- Kornman and colleagues (2000) noted that we
Probing depth increases
tion. This is especially true in complex periodon- must not confuse risk potential by basing future
Bleeding on probing
tal prosthetic cases in which treatment decisions prognosis on the current diagnostic assessment: Suppuration
are based predominantly on subjective factors. “For unlike ‘diagnosis’ that looks at ‘what is,’ Level of plaque control
The modern paradigm for periodontal prognosis ‘prognosis’ determines ‘what may become’ of the Low socioeconomic level
can be seen in Figure 1-1. disease” (Figure 1-2). Sporadic dental care
In discussing prognosis, we must first differ- Historically, the pathogenesis of human Level of education
entiate among diagnosis, risk, and prognosis: periodontal disease was described by Page and Poor dietary habits
Schroeder (1976). It was agreed that the disease Infectious and other acquired diseases
1. Diagnostic factors: factors associated with the was initiated and perpetuated by a small group of Side effects of medication
analysis and determination of a disease process gram-negative anaerobic or microaerophilic bac- Adapted from Newman and Kornman (1994).
2. Risk factors: factors associated with dis- teria that form subgingival colonies (Page and
ease development in people who do not yet Kornman, 1997). The process was thought to be
have the disease (Table 1-1) (Newman and continuous in nature (continuous theory) until it of periodontal progression, which assumed all
Kornman, 1994) was shown to occur episodically or in random plaque to be similar, with equal susceptibility for
3. Prognostic factors: factors used to predict bursts (random burst theory) (Haffajee and col- everyone (Figure 1-3) (McGuire, 2000).
disease progression once the disease is pre- leagues, 1983; Haffajee and Socransky 1996). The Clinical paradigms for prognosis were then
sent (Table 1-2) (McGuire and Nunn, 1996a) continuous theory resulted in a simplistic model based predominantly on environmental or
anatomic factors that limited or increased plaque
Significant decrease in Less favorable
prognosis and prognosis with
increase in tooth loss increase in tooth loss
Table 1-2 Commonly Taught Clinical Factors
Used in Assigning Prognosis
+ +
Heavy Heavy
Smoking stopped Smoking stopped
smoking smoking Individual tooth prognosis
Percentage of bone loss
++
Deepest probing depth (in mm)
Can be successfully IL-1 IL-1 Favorable long term
POSITIVE
Patient
prognosis
Horizontal or vertical bone loss
treated and maintained NEGATIVE
even though they may Deepest furcation involvement: 0, 1, 2, 3
have greater tooth loss Mobility: 0, 1, 2, 3
Host systemic Host systemic
Crown-to-root ratio: favorable or unfavorable
factor negative factor negative Root form: favorable or unfavorable
Caries or pulpal involvement: yes or no
ENVIRONMENTAL FACTORS ENVIRONMENTAL FACTORS Tooth malposition: yes or no
*Mobility
**Occlusal
*Mobility Fixed or removable abutment: yes or no
Unfavorable crown-root ratio Unfavorable crown-root ratio
Percentage of bone loss discrepancies Percentage of bone loss
Initial bone level Initial bone level Overall prognosis
Furcation involvement Furcation involvement Age
Probing depth Probing depth Significant medical history (smoker and/or
Plaque control Plaque control
Parafunction without night guard Parafunction without night guard
diabetic)
Family history of periodontal disease (mother,
Significant decrease in A catalyst for A reliable methodology father, sibling): yes or no and whom
prognosis and increase periodontal for selecting teeth with Hygiene: good, fair, poor
in tooth loss breakdown a "good" prognosis
but “less” accurate for Compliant: yes or no
predicting the prognosis Maintenance interval: 2 months, 2 months
NOTE of teeth with a “less than alternate, 3 months, 3 months alternate
* Any factor that increased mobility decreases prognosis good” prognosis
** Untreated occlusal discrepancies are always significant Parafunctional habit with bite guard
+ Smoking is always a negative modifying factor Parafunctional habit without bite guard
++ Genotype appears to be significant in untreated or poorly maintained patients
Adapted from McGuire and Newman (1996).
FIGURE 1-1. Periodontal paradigm for determining prognosis.
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2 Basics
Good prognosis (one or more of the following): control of the etiologic factors and adequate periodontal
Table 1-4 Clinical Parameters
support as measured clinically and radiographically to ensure that the tooth would be relatively easy to
maintain by the patient and clinician, assuming proper maintenance Mobility*†
Fair prognosis (one or more of the following): approximately 25% attachment loss as measured clinically Furcation involvement (severe)†
and radiographically and/or Class I furcation involvement. The location and depth of the furcation would Probing depth†
allow proper maintenance with good patient compliance. Unfavorable crown-to-root ratio
Questionable prognosis (one or more of the following): 50% attachment loss with Class II furcations. The Percent bone loss*†
location and depth of the furcations may limit proper maintenance. Parafunctional habit without a night guard
Malposed tooth
Poor prognosis (one or more of the following): greater than 50% attachment loss resulting in a poor crown- Smoking
to-root ratio; poor root form; Class II furcations not easily accessible to maintenance of Class III furcations;
2+ mobility or greater; significant root proximity Plaque control (compromised teeth tend not to
get worse under maintenance)*†
Hopeless prognosis: inadequate attachments to maintain the tooth; extraction performed or suggested
*Consistent with Ghiai and Bissada (1994).
†
Adapted from McGuire and Nunn (1996). Consistent with Nieri and colleagues (2002).
Cohen_001-008_01.qxd 11/16/06 8:19 PM Page 3
Prognosis 3
Cumulative % of subjects
with ≥ 30% mean boneloss
35 Genotype Pos
Severe N=18
N=36
30
Genotype Neg
Moderate N=37 25 N=63
20
15
Mild N=44
10
0 10 20 30 40 50 60 70 80 5
0
Percentage of subjects who were genotype positive 35-40 41-45 46-50 51-55 56-60 >60
(IL-1A allele 2 plus IL-1B+3953 allele 2) Age
FIGURE 1-5. The occurrence of the composite genotype for nonsmok- FIGURE 1-6. The cumulative frequency distribution of nonsmokers with severe bone
ers in different disease groups. (Adapted from K Kornman et al. J Clini- loss (≥ 30%) at different age groups. (Adapted from Kornman et al, J Clinical Periodon-
cal Periodontology 1997) tology, 1997)
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4 Basics
Prognosis 5
No. of teeth
most significant determinates. Younger patients,
0.6
n=28 although possibly possessing greater adaptability
0.5
0.4 n=101 and regenerative function, must retain their teeth
0.3 n=125 significantly longer than older patients. The peri-
0.2 odontal destruction observed in the elderly is one
0.1 of lifetime disease accumulation and not an age-
0 specific condition (American Academy of Perio-
Nonsmokers Smokers dontology, 1995). This disease accumulation pro-
PST + PST - vides us with a longer history of their
FIGURE 1-8. The mean number of lost teeth per subject per 10 years
in smokers and nonsmokers testing positive (PST+) negative (PST-) for
genetic polymorphism of interleukin. (Adapted from Ayelson et al. Diag-
nosis and Risk Prediction of Periodontal Disease, 2002. Quintessence
, Illinois)
They also found Note: Similar results were seen in patients with
1. No relationship to furcation involvement good oral hygiene, bringing into question the need
2. In patients with good oral hygiene, OD was for significant etiologic factors for occlusion to
the only significant predictor of increased prob- have a negative impact.
ing depth and initial unfavorable prognosis.
Harrel and Nunn concluded: “Based on the
3. Although cofactors such as smoking (increased
results obtained in this study, there is evidence
pocketing), gender (men had deeper probing
that untreated occlusal trauma is certainly a cat-
depths than women), and oral hygiene
alyst for the progression of periodontal disease.”
(patients with good hygiene had less probing
Harrel (2003) noted that occlusion should be
depth than patients with poor hygiene) had
considered a cumulative risk factor, similar to
an impact, there was no “casual pathway”
smoking, that should be (Hallmon and Harrel
(Kornman and colleagues, 1997; McGuire
2004) part of routine periodontal treatment.
and Nunn, 1999) and patients with OD still
had a 1 mm greater probing depth. Modifying Factors A
B. Post-treatment findings Anterior Eshtetics. The modern periodontal
Teeth with OD had significantly greater paradigm is predicated on papillary preservation
1. Likelihood of a worsening prognosis maintainable on gingival esthetics and the inter-
2. Risk of increased mobility relationship of the lip, gingival, and occlusal lines.
3. Increase in probing depth (Figures 1-12 Therefore in the esthetic zone, the clinician must
and 1-13) first evaluate all treatments on two primary con-
80
SMO-PST- SMO-PST+
70
60 SMO+PST- SMO+PST+
Patient (%)
50
40
30
20
10
0
0 1 2 3-4 5-7 8+ B
Number of lost teeth
FIGURE 1-10. Silastic model displaying,
FIGURE 1-9. Frequency distribution of lost teeth in nonsmokers testing A, Even force distribution of force applied
positive (SMO-PST+), smokers testing negative (SMO+PST-) and smokers in long axis of tooth; B, Uneven cervical
testing positive (SMO+PST+) for genetic polymorphism of interleukin-1. and apical distribution of forces when an
(Adapted from Ayelson et al. Diagnosis and Risk Prediction of Periodontal angular force is applied. (Courtesy of Dr.
Disease, 2002. Quintessence, Illinois) Irving Glickman.)
Cohen_001-008_01.qxd 11/16/06 8:19 PM Page 6
6 Basics
periodontal, functional, and restorative status, colleagues, 1984; Neuman and colleagues, 1994; with the prognosis for proper triage. They break
offering greater diagnostic insight and thus Nieri and colleagues, 2002). down their diagnostic parameters for risk and
enhancing our ability to determine or predict prognosis assessment into the following:
Clinical Skill Level and Knowledge. Modern
how their teeth may continue to function in the
periodontal therapy has become a highly com- 1. Periodontal
future. What might not be acceptable in a young
plex specialty requiring a diversity of skills, which 2. Biomechanical
person might be acceptable in an older patient.
come only with time, experience, continuing a. Caries susceptibility
Motivation, Cooperation, and Level of Plaque education, and personal application. It is difficult b. Extent of structural compromise
Control. Plaque control is the single most impor- to be an expert in all phases of therapy. Our per- 3. Functional
tant factor in the treatment of periodontal disease. sonal skill limitations should not restrict our a. Temporomandibular joint disorders
The long-term success of any case is predicated patients’ treatment options or their success. We b. Mobility
on a patient’s ability to maintain an adequate must therefore recognize our limitations and 4. Dentofacial
level of plaque control. This is even more so in refer our patients to someone who possesses a
Dentofacial risk is based on the degree of
advanced periodontal prosthetic cases. Complex greater degree of competence in a particular area.
tooth display and our ability to achieve ideal
advanced periodontal and or prosthetic cases
tooth position in relationship to the face.
require a highly motivated and cooperative Risk of Treatment
patient for successful resolution. Plaque control,
Barkman and Kois (2005) stated that the risk of Note: Barkman and Kois (2005) make the point
motivation, and cooperation must therefore be
treatment must be determined and combined that as the risk of treatment increases and prog-
carefully evaluated prior to starting (Becker and
nosis decreases, treatment begins to move toward
implant-supported restorations.
2.0
3.0 Summary
Changing probing depth (mm)
1.5
Changing probing depth (mm)
Prognosis 7
d. Increased probing depth 6. IL-1-positive smokers should be regarded as Note: Kaldahl (1996) studied the relationship
e. Greater initial bone loss potential higher-risk patients, whereas IL-1- between periodontal treatment and smoking and
4. Genotype is a prediction of potential future negative nonsmokers are considered at low concluded that “while the negative effects of
risk in untreated periodontal cases. risk of developing tooth loss and bone loss. smoking on therapy were sustained or increased
5. Patients who are IL-1 positive can be treated 7. Smokers can be treated successfully if they over time, it must be remembered that periodontal
successfully, be well maintained over the will cease smoking. therapy in all groups produced improvements of all
years, and do not require altered treatment 8. In the presence of good plaque control, long- clinical parameters.”
modalities. term success is improved and risk factor influ-
ence is diminished except when ODs are present.
Continued
Cohen_001-008_01.qxd 11/16/06 8:19 PM Page 8
8 Basics
IL = interleukin.
Conclusion ratio of 51.9). This means that all of these cases, with a good or good to fair prognosis should be
irrespective of genotype, smoking, and/or clinical used as abutments and ODs should be eliminat-
It is important to note that of the 2,610 teeth (102 ed. Finally, it should always be remembered that a
parameters, can be successfully treated over a long
patients) followed in the studies by Nieri and col- highly motivated and cooperative patient can
period of time (10–14 years). In complex peri-
leagues (2002) and McGuire and Nunn (1999), very often overcome many if not all negative fac-
odontal-prosthetic cases involving patients with a
only 99 were lost (3.8%). Most of these teeth had tors and that the converse is also true (Table 1-7).
positive genotype and/or smokers, only teeth
an original prognosis of poor or hopeless (risk
Cohen_009-014_02.qxd 11/16/06 8:21 PM Page 9
Surgical Basics
Basic Incisions Classification of Surgical Procedures Increasing Dimension of
Exisiting Attached Gingiva.
Peridontal disease is multifaceted in the nature, Correction of Soft Tissue Pockets 1. Mucosal stripping
scope, and types of problems created (eg, 2. Periosteal separation
Closed Procedures.
mucogingival problems, osseous deformities, 3. Laterally positioned flap (pedicle)
1. Curettage
gingival enlargement); therefore, many types of a. Full thickness
2. Excisional new attachment procedure (ENAP)
treatment exist (Figure 2-1). There is no one way b. Partial thickness
and modified ENAP
to approach a single problem or procedure. Train- c. Periosteally stimulated
3. Modified Widman flap
ing, ability, philosophy, and objectives ultimately d. Partial/full thickness
4. Apically positioned (repositioned) flap
determine final treatment selection. The follow- 4. Papillary flaps
a. Full thickness
ing is a list of basic surgical incisions. a. Double papillae
b. Partial/full thickness
1. Curettage: The removal of the inner epithe- c. Partial thickness (supraperiosteal) b. Rotated papillae
lial lining, epithelial attachment, and under- 5. Palatal flap c. Horizontal papillae
lying inflamed connective tissue on the inner a. Full thickness 5. Edlan-Mejchar, subperiosteal vestibular
aspect of the pocket. This is a closed surgical b. Partial thickness extension operation, or double lateral bridg-
procedure (Figure 2-2A). 6. Distal wedge procedure ing flap
2. Gingivectomy: The excisional removal of tis- a. Tuberosity 6. Free soft tissue autografts
sue for treatment of suprabony pockets. This b. Retromolar area a. Partial thickness
procedure is indicated where bone loss is b. Full thickness
Open Procedures. 7. Connective tissue autograft
horizontal and there is an adequate zone of 1. Gingivectomy
attached keratinized gingiva (Figure 2-2B). 8. Subepithelial connective tissue graft
2. Gingivoplasty
3. Full-thickness (mucoperiosteal) flap: A flap
designed to gain access and visibility for Procedures Commonly Used
Surgery for Correction of Osseous for Root Coverage
osseous surgery, relocation of the frenulum, Deformities and Osseous Enhancement
maintenance of the attached tissue, and Procedures Pedicle Flaps (Full or Partial Thickness).
pocket elimination and regeneration proce- 1. Laterally positioned flaps
dures. The incision can be sulcular, crestal, or Closed Procedures. 2. Double-papillae flaps
inverse bevel, depending on the amount of 1. Full- or partial-thickness flap 3. Coronally positioned flaps
attached tissue present (Figure 2-2C). a. Apically positioned flap 4. Periosteally stimulated flaps
4. Partial- or split-thickness (mucosal) flap: A b. Unpositioned flap 5. Semilunar flap
flap designed to retain and maintain the c. Modified flap 6. Rotated or transpositional pedicle flap
periosteal covering over the bone. A sharp or d. Modified Widman flap
supraperiosteal dissection technique parallel 2. Distal wedge procedure Free Soft Tissue Autografts.
to the bone is used in this procedure. It is indi- 3. Palatal flap 1. Full thickness
cated mostly in areas of thin bony plates and 2. Partial thickness
Open Procedures.
for mucogingival procedures (Figure 2-2D). 1. Gingivectomy Subepithelial Connective Tissue Graft.
5. Modified full-thickness (mucoperiosteal) flap: a. Rotary abrasives
A flap for which a first-stage gingivectomy Acellular Dermal Matrix Grafts.
b. Interproximal denudation
incision is used for pocket reduction or elimi- c. Intrabony pocket procedure Guided Tissue Regeneration.
nation, followed by a secondary inverse- 2. Prichard procedure for osseous fill 1. Nonresorbable
beveled incision to the crest of bone. This tech- 2. Resorbable
nique requires an adequate zone of attached Guided Tissue Regeneration (GTR).
keratinized gingiva and is used primarily on Guided Bone Regeneration (GBR). Procedure Commonly Used
the palate, on enlarged tissue, or in areas in for Ridge Augmentation
which limited access may prevent a primary Correction of Mucogingival Problems
inverse-beveled incision (Figure 2-2E). Connective Tissue Graft.
Preservation of Existing Attached Gingiva. 1. Pouch procedure
Tables 2-1 and 2-2 compare the various 1. Apically positioned (repositioned) flap 2. Connective tissue graft/coronally positioned
treatment procedures. These should be used only a. Full thickness flap
as a general guide in deciding which technique to b. Partial thickness 3. Pediculated connective tissue graft
use. Table 2-3 is a comparative analysis of the var- 2. Frenectomy or frenotomy 4. Onlay interpositional graft
ious surgical techniques. 3. Modified Widman flap 5. Interpositional graft
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10 Basics
Historical review
Radical gingivectomy flap procedure
Unrepositioned flap 1862 - 1884 Apically displaced flap
Cizezinky 1914 S.Robicsek Neuman 1912
Coronally Bentler 1916 Widman 1916
repositioned flap
Norberg 1926
Semiflap All bone healthy Repositioned flap
Kirkland 1931 (Not necrotic) Zemsky 1926
Kronfeld 1935
Modified flap
Kirkland 1936
Treatment of
pre-maxilla
Ingle 1952
Classification of
intrabony defects
Apical repositioned split-flap Split thickness Use of two 1958
Stewart 1960 lat. flap vertical incisions
1954 1964 1957
Autogenous
Periosteal bone chips
separation From edentulous The apically Palatal approach 1964
1961, 1962 ridge repositioned flap for osseous surgery
Obwegeser 1964 1962 1963, 1964
1956
Demineralized freeze
Double flap dried allografic bone
1963 Double lat. Grupe Classification (DFDAB) 1965, 1968
reposit. flap modification of flaps
1963 1966 1964
Fixed long labial Bone swaging
Elden - Mejchar
mucosal flap 1965
1963
1963 Oblique rotated Contiguous lat. Unrepositioned
Palatal ledge and
Flap sliding flap flap
wedge technique
1965 1967 1965
1958, 1965 Illac crest
Free gingival graft
bone implants
1966
1968
Horizontal sliding For furcation The distal wedge
Cosmetic root Ridge papillary flap involvement 1963, 1964,
coverage augmentation 1967 1968 1966
Bone from
extraction sites
1969
Classification Classification Double papillae Periosteal- Curtain Split-thickness
1968, 1985 1983 flap stimulated flap procedure palatal flap
1968 1968 1969 1969
Osseous
S.V.E. coagulum
1976 Coronally positioned flap Roll technique 1970
1976, 1986 1979 Rotated lat. Gingival fiber
sliding flap retention
1969 1972
Free gingival grafts Onlay grafting Tuberosity
1982, 1985 1979, 1983 grafts
1971
Modified Widman
flap
Subepithelial C.T. graft Subepithelial C.T. graft 1974
Double lat. 1985, 1986 1979, 1982 Bone
bridging fap blending
1985 1972
Envelope flap Pouch procedure E.N.A.P.
1985 1980, 1981 1976
Surgical Basics 11
C D E
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12 Basics
Table 2-1 Comparison of Open (Gingivectomy) versus Closed (Flap) Procedures Surgical Considerations
Open Closed (Partial- or 1. Procedural selection should be based on the
Variables (Gingivectomy) Full-Thickness Flaps) following:
Healing Secondary intention Primary intention a. Simplicity
Time requirement for completion of procedure Fast Slower b. Predictability
Reattachment No Possible c. Efficiency
Degree of difficulty Low High d. Mucogingival considerations
Bleeding postoperatively Yes Minimum e. Underlying osseous topography
Visibility for osseous surgery Inadequate Good f. Anatomic and physical limitations (eg,
Ability to treat irregularities and defects Inadequate Good small mouth, gagging, mental foramen)
Preservation of keratinized gingiva No Yes
g. Age and systemic factors (eg, cardiac
arrhythmias and murmurs, diabetes, his-
tory of radiation treatment, hypothy-
Procedures Commonly Used for Socket hemorrhagic disorders) should be under roidism, hyperthyroidism)
Preservation adequate control. Medications should be 2. All incisions should be clear, smooth, and
carefully noted, and medical consultations denifite. Indecision usually results in an
1. Basic procedure
and preoperative laboratory work should be uneven, ragged incision, which requires more
a. Socket filler
performed where indicated. It is important healing time.
b. Connective tissue graft
to note that the medical history consists of 3. All flaps should be designed for maximum
2. Socket seal
a review of drug abuse, transfusion, and use and retention of keratinized gingival tis-
3. CollaPlug (Sulzer Medica, Carlsbad, Cali-
alternative lifestyles in attempting to deter- sue so as to maintain a functional zone of
fornia)
mine the risk of acquired immune deficien- attached keratinized gingiva and prevent
4. Prosthetic support
cy syndrome (AIDS) or human immunode- needless secondary procedures.
ficiency virus (HIV). This should be 4. The flap design should allow for adequate
Procedures Commonly Used combined with a thorough oral examination access and visibility.
for Papillary Reconstruction (eg, ulcers, candidiasis, hairy leukoplakia). 5. Involvement of adjacent noninvolved areas
1. Connective tissue grafts Note: The best protection against AIDS and should be avoided.
2. Bone graft/connective tissue graft hepatitis is a proper barrier technique and 6. The flap design should prevent unnecessary
sterilization at all times. bone exposure, with resultant possible loss
Contraindications for Periodontal 2. Blood pressure should be recorded. and dehiscence or fenestration formation.
Surgery (Lindhe, 2003) 3. Surgical therapy should be considered only 7. Where possible, primary intention procedures
1. Patient cooperation after adequate control, scaling, root planing, are preferred to those of secondary intention.
2. Cardiovascular disease and all necessary restorative, prosthetic, 8. The base of a flap should be as wide as
a. Uncontrolled hypertension endodontic, orthodontic, and occlusal stabi- the coronal aspect to allow for adequate
b. Angina pecton’s lization and splinting procedures have been vascularity.
c. Myocardial infarction completed and the case has been reevaluat- 9. Tissue tags should be removed to allow for
d. Anticoagulant therapy ed. Without proper plaque control, there is no rapid healing and prevent regrowth of gran-
e. Rheumatic endocarditis, congenital heart need for surgery. ulation tissue.
lesions, and heart vascular implants 4. A surgical consent form should be complet- 10. Adequate flap stabilization is necessary to
3. Organ transplants ed in all cases, and periodontal documenta- prevent displacement, unnecessary bleeding,
4. Blood disorders tion (including tissue quality, pocket depths, hematoma formation, bone exposure, and
5. Hormonal disorders radiographs, and models) is a must. possible infection.
a. Uncontrolled diabetes
b. Adrenal dysfunction
6. Hematologic disorders
Table 2-2 Comparison of Full- and Partial-Thickness Flaps
a. Multiple sclerosis and Parkinson’s disease
b. Epilepsy Full Thickness Partial Thickness
7. Smoking—more a limiting factor than a Variables (Mucoperiosteal) (Mucosal)
contraindication Healing Primary intention Secondary intention
Degree of difficulty Moderate High
Note: No periodontal surgery should be undertaken Pocket elimination Yes Yes
on a medically compromised patient without a recent Osseous surgery, resective or inductive Yes No
physical evaluation and clearance by a physician. Periosteal retention No Yes
Relocation of frenum Yes Yes
Widen zone of keratinized gingiva No Yes
General Surgical Considerations
Increase in attached keratinized gingiva Yes Yes
Presurgical Considerations Combine with other mucogingival procedures No Yes
Suture variability Low High
1. A complete medical history should be taken
Presence of a thin periodontium—dehiscence or fenestration No Yes
and any underlying systemic disorders or
Bleeding and tissue trauma Limited Greater
problems (ie, hypertension, diabetes, or
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Surgical Basics 13
I II III
Adapted from Kinoshita S, Wen RC. Color atlas of periodontics. St. Louis: Mosby-Year Book; 1985.
ENAP = excisional new attachment procedure.
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18 Basics
Principles of Suturing
Ethicon (1985) recommends the following prin-
ciples for knot tying:
1. The completed knot must be tight, firm, and
tied so that slippage will not occur.
2. To avoid wicking of bacteria, knots should
not be placed in incision lines.
3. Knots should be small and the ends cut short
(2–3 mm).
4. Avoid excessive tension to finer-gauge mate-
rials because breakage may occur.
5. Avoid using a jerking motion, which may
break the suture.
6. Avoid crushing or crimping of suture mate-
rials by not using hemostats or needle hold-
ers on them except on the free end for tying.
7. Do not tie the suture too tightly because tis-
sue necrosis may occur. Knot tension should
not produce tissue blanching. FIGURE 3-2. Suturing knots.
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Surgical Needles
Most surgical needles are fabricated from heat-
treated steel and possess a microsilicon finish to
diminish tissue drag and a tip that is extremely
sharp and has undergone electropolishing
(Ethicon, 1985). The surgical needle has a basic
design composed of three parts (Figure 3-3):
1. The eye which is press-fitted or swaged (eye-
less) permits the suture and needle to act as
a single unit to decrease trauma.
2. The body which is the widest point of needle
and is also referred to as the grasping area. The
body comes in a number of shapes (round,
oval, rectangular, trapezoid, or side flattened).
3. The point which runs from the tip of the
maximum cross-sectional area of the body. FIGURE 3-3. Needle anatomy. Needles are described by their arc. Most periodontal surgical needles are of
The point also comes in a number of differ- three-eighths or one-half curvature. Different components of the needle are described.
ent shapes (conventional cutting, reverse
cutting, side cutting, taper cut, taper, blunt)
(Figure 3-4).
4. The chord length is the straight line distance Placement of Needle in Tissue Suturing Techniques
between the point of a curved needle and the
Ethicon (1985) gives the following principles for Different suturing techniques may employ either
swage.
placing the needle in tissue: periosteal or nonperiosteal suture placement:
5. The radius is the distance measured from the
center of the circle to the body of the needle 1. Force should always be applied in the direc-
1. Interrupted
if the curvature of the needle was continued tion that follows the curvature of the needle.
a. Figure eight
to make a full circle. 2. Suturing should always be from movable to
b. Circumferential director loop
nonmovable tissue.
c. Mattress—vertical or horizontal
Needle Holder Selection 3. Avoid excessive tissue bites with small needles
d. Intrapapillary
because it will be difficult to retrieve them.
Ethicon (1985) gives the following pointers for 2. Continuous
4. Use only sharp needles with minimal force.
selecting a needle holder: a. Papillary sling
Replace dull needles.
b. Vertical mattress
1. Use an approximate size for the given needle. 5. Grasp the needle in the body one-quarter to
c. Locking
The smaller the needle, the smaller the nee- half the length from the swaged area. Do not
dle holder required. hold the swaged area; this may bend or break The choice of technique is generally made on
2. The needle should be grasped one-quarter to the needle. Do not grasp the point area the basis of a combination of the individual oper-
half the distance from the swaged area to the because damage or notching may result (see ator’s preference, educational background, and
point, as shown in Figure 3-5. Figure 3-5). skill level, as well as surgical requirements.
3. The tips of the jaws of the needle holder a. Prior to suturing, the needle holder is
should meet before the remaining portions repositioned to the forward half of the
of the jaws. needle with a few millimeters of the tip,
Periosteal Suturing
4. The needle should be placed securely in the tips as shown in Figure 3-5. Periosteal suturing generally requires a high
of the jaws and should not rock, twist, or turn. 6. The needle should always penetrate the tis- degree of dexterity in both flap management and
5. Do not overclose the needle holder. It sue at right angles. suture placement. Small needles (P-3), fine
should close only to the first or second a. Never force the needle through the tissue. sutures (4-0 to 6-0), and proper needle holders
rachet. This will avoid damaging or notch- 7. Avoid retrieving the needle from the tissue are a basic requirement. Periosteal suturing per-
ing the needle. by the tip. This will damage or dull the nee- mits precise flap placement and stabilization.
6. Pass the needle holder so that it is always dle. Attempt to grasp the body as far back
directed by the surgeon’s thumb. as possible. Technique
7. Do not use digital pressure on the tissue; this 9. An adequate tissue bite (≥ 2–3 mm) is The five steps here are used in periosteal suturing
may puncture a glove. required to prevent the flap from tearing. (Chaiken, 1977) and are seen in Figure 3-6:
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20 Basics
FIGURE 3-4. Cutting needles. Both outline and cross-sectional views of the various forms of cutting needle are shown. Conventional
cutting and reverse cutting are also shown.
A A'
B B'
C C'
FIGURE 3-5. Correct handling of suture needles. A, Needle holder holding a suture needle just anterior to
the curvature; correct position; A', suture needle undamaged. B, Suture needle held incorrectly at tip; B', tip
of suture needle damaged. C, Suture needle held incorrectly behind curvature; C', needle bent as a result.
Cohen_015-028_03.qxd 11/16/06 8:23 PM Page 21
1. Penetration: The needle point is positioned tightly against the bone so as not to damage resulting in lifting or tearing of the perios-
perpendicular (90°) to the tissue surface and or dull the needle point (see Figure 3-6B). teum (see Figure 3-6, D and E).
underlying bone. It is then inserted com- 3. Glide: The needle point is now permitted to 5. Exit: The final stage of gliding and rotation is
pletely through the tissue until the bone is glide against the bone for only a short dis- needle exit. The needle is made to exit the
engaged. This is as opposed to the usual 30° tance. Care must be taken not to lift or dam- tissue through the gentle application of pres-
needle insertion angle (see Figure 3-6A). age the periosteum (see Figure 3-6C). sure from above, thus allowing the tip to
2. Rotation: The body of the needle is now 4. Rotation: As the needle glides against the pierce the tissue. If digital pressure is to be
rotated about the needle point in the direc- bone, it is rotated about the body, following used, care must be used to avoid personal
tion opposite to that in which the needle is its circumferenced outline. In this way, the injury (see Figure 3-6F).
intended to travel. The needle point is held needle will not be pushed through the tissue,
A B C
D E F
FIGURE 3-6. Periosteal suturing. A, Needle penetration; needle point is perpendicular to bone. B, Rotation of needle body about point.
C,D, The needle is moved along the bone below the periostum. E, Rotation about needle body permitting point to exit periosteum and
tissue. F, Completed periosteal suture.
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22 Basics
Interrupted Sutures Technique sure while avoiding suture contact with the implant
material, thus preventing wicking. They are left for
Indications Figure Eight and Circumferential Sutures. 14 to 21 days (Mejias, 1983) and therefore require
Interrupted sutures are most often used for the Suturing is begun on the buccal surface 3 to 4 mm a suitable material (eg, nylon, e-PTFE) that is
following: from the tip of the papilla to prevent tearing of the biologically inert and does not rapidly “wick.”
thinned papilla. The needle is first inserted into
1. Vertical incision
the outer surface of the buccal flap and then either Vertical Mattress Technique. The flap is stabi-
2. Tuberosity and retromolar areas
through the outer epithelialized surface (figure lized and a P-3 needle is inserted 7 to 10 mm api-
3. Bone regeneration procedures with or with-
eight) (Figure 3-8) or the connective tissue under cal to the tip of the papilla. It is passed through the
out guided tissue regeneration
the surface (circumferential) (see Figure 3-8A) of periosteum (if periosteal sutures are being used),
4. Widman flaps, open flap curettage, unreposi-
the lingual flap. The needle is then returned through emerging again from the epithelialized surface of
tioned flaps, or apically positioned flaps where
the embrasure and tied buccally. the flap 2 to 3 mm from the tip of the papilla. The
maximum interproximal coverage is required
When interproximal closure is critical, the needle is brought through the embrasure, where
5. Edentulous areas
circumferential suture will permit greater coapta- the technique is again repeated lingually or palatal-
6. Partial- or split-thickness flaps
tion and tucking down of the papilla because of ly. The suture is then tied buccally (Figure 3-9A).
7. Osseointegrated implants
the lack of intervening suture material between
Types the tips of the papilla. Horizontal Mattress Technique. A P-3 needle is
inserted 7 to 8 mm apical to and to one side of the
In Figure 3-7, we see the four most commonly Mattress Sutures. Mattress sutures are used for midline of the papilla, emerging again 4 to 5 mm
used interrupted sutures: greater flap security and control; they permit through the epithelialized surface on the oppos-
1. Circumferential, direct, or loop (see Figure more precise flap placement, especially when ing side of the midline (Figure 3-9B). The suture
3-7A) combined with periosteal stabilization. They also may or may not be brought through the perios-
2. Figure eight (see Figure 3-7B) allow for good papillary stabilization and place- teum. The needle is then passed through the
3. Vertical or horizontal mattress (see Figure ment. The vertical mattress (nonperiosteal) suture embrasure, and the suture, after being repeated
3-7C) is recommended for use with bone regeneration lingually or palatally, is tied buccally. For greater
4. Intrapapillary placement (see Figure 3-7D) procedures because it permits maximum tissue clo- papillary stability and control, the double parallel
A B
C D
FIGURE 3–7. Four interrupted sutures. A, Circumferential. B, Figure-eight. C, Vertical mattress. D, Intrapapillary.
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A B A B
FIGURE 3-8. A, Circumferential suture. B, Figure-eight suture. FIGURE 3-9. A, Horizontal mattress suture. B, Vertical mattress suture.
A B C
D E F
FIGURE 3-10. Sling suture about adjacent tooth.
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24 Basics
strands of this suture can be made to cross over predominantly when standard interproximal inci- sion and above the first suture (Figure 3-13A2).
the three tops of the papillae. This is the double sions are used. Start bucally below the papilla (2–4 The suture is passed through the interproximal
crossed-over suture. mm) and insert the needle to and then through papilla and returned as a horizontal mattress
the undersurface of the lingual flap (Figure 3- suture on the buccal surface and tied off.
Intrapapillary Placement. This technique is rec-
12A1). The suture needle is then reinserted lin-
ommended for use only with modified Widman
gually 2–4 mm above the initial suture and contin- Retromolar Suture Modification for Primary Cov-
flaps and regeneration procedures in which there is
ued to and then through the buccal flap (Figure erage. This technique (Hutchenson 2005) (Fig-
adequate thickness of the papillary tissue.
3-12A2). The suture is then brought lingually over ure 3-14) is specially designed for gaining inti-
A P-3 needle is inserted buccally 4 to 5 mm
the coronal aspect of the flap and through the loop mate tissue-tooth contact where regeneration is
from the tip of the papilla and passed through the
(Figure 3-12A3). The suture is afterwards returned being attempted. It is employed when there is an
tissue, emerging from the very tip of the papilla.
bucally and sutured (Figure 3-12A4). Figure 3-12B intrabony defect distal to the last tooth on the
This is repeated lingually and tied buccally, thus
shows the completed suture. lower teeth. It not only permits primary flap clo-
permitting exact tip-to-tip placement of the flaps
sure but close approximation of the tissue on the
(see Figure 3-7D). Modified Flap Suturing Technique. This tech-
distal aspect of the tooth. Figure 3-14A shows a
nique (Cortellini et al 1995) was introduced
Sling Suture. The sling suture is primarily used defect distal to the last tooth. The arrows on Fig-
specifically for achieving maximum interproxi-
for a flap that has been raised on only one side of ure 3-14B indicates desired movement of flap and
mal coverage and primary closure over intrabony
a tooth, involving only one or two adjacent papil- dotted lines indicate ideal flap position. Flap ide-
defect is treated by GTR. The modified flap tech-
lae. It is most often used in coronally and lateral- ally should be positioned against distal surface of
nique (Figure 3-13) requires the initial incision
ly positioned flaps. The technique involves use of tooth with primary closure. Suture is begun on
be made at the buccal line angles in the area of
one of the interrupted sutures, which is either the mesiobuccal of the terminal tooth (Figure
the interproximal defect. It is a papillary preser-
anchored about the adjacent tooth (Figure 3-10) 3-14C1). The suture is passed through interprox-
vation technique. The suturing permits coronal
or slung around the tooth to hold both papillae imal to the distal and inserted through only the
positioning, flap stabilization, and primary inter-
(Figure 3-11). undersurface of the buccal flap. The suture is
proximal closure. The first suture is begun buc-
brought almost 360° around the tooth starting
Specialized Interrupted Suturing Techniques for cally 5–6 mm below the initial incision (Fig-
lingually and continuing bucally until again
Bone Regeneration and Retromolar and Tuberos- ure 3-13A1). The suture is passed through the
reaching the distal surface (Figure 3-14C2). The
ity Areas. Laurell Modification. Laurell modi- buccal and palatal flaps. It is then reinserted
needle is passed through the undersurface of the
fied mattress suture (1993) (Figure 3-12) for coro- palatally and allowed to exit the buccal flap 2 mm
lingual flap and tied on the buccal surface (Figure
nal flap positioning and primary flap coverage is a above the initial placements. This is tied off and
3-14C3). Figure 13-14D shows suturing having
technique which, although capable of being should stabilize the body of the flap. The second
been completed and primary coverage attained.
employed for all regenerative techniques, is used suture is now begun 3–4 mm below the initial inci-
A B C
D E F
FIGURE 3-11. Sling suture about single tooth.
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FIGURE 3-12. Laurell modified mattress suture (see text). FIGURE 3-13. Modified flap suturing technique (see text).
Continuous Sutures Sling loose or the suture may come untied from multi- interproximal embrasure (see Figure 3-15B) in
ple teeth. such a manner that the suture is made to encircle
When multiple teeth are involved, the continuous the neck of the tooth (see Figure 3-15C). The
suture is preferred. Types needle is then passed either over the papilla and
The choice of continuous suture depends on the through the outer epithelialized surface or under-
Advantages operator’s preference. These, too, can be periosteal neath and through the connective tissue under-
1. Can include as many teeth as required or nonperiosteal: surface of the papilla. The needle is passed again
2. Minimizes the need for multiple knots through the embrasure and continued anteriorly
1. Independent sling suture
3. Simplicity (Figure 3-15D). This procedure is repeated
2. Mattress sutures
4. The teeth are used to anchor the flap through each successive embrasure until all
a. Vertical
5. Permits precise flap placement papillae have been engaged.
b. Horizontal
6. Avoids the need for periosteal sutures
3. Continuous locking
7. Allows independent placement and tension Note: For maximum flap control, it is best to pass
of buccal and lingual or palatal flaps. Buccal the needle through the connective tissue under-
flaps can be positioned loosely, whereas lin-
Technique surface of the papilla.
gual and palatal flaps are pulled more tightly Independent Sling Suture
about the teeth. The continuous sling suture (Figure 3-15), A terminal end loop (Figure 3-15E) is then
8. Greater distribution of forces on the flaps although most often begun as a continuation of used if a single flap has been reflected or if the
tuberosity or retromolar suturing (see Figure flaps are to be sutured independently. In this
Disadvantages 3-15A), can also be started with a looped suture manner, the flaps are tied against the teeth as
The main disadvantage of continuous sutures is about the terminal papilla (buccal, lingual, or opposed to each other.
that if the suture breaks, the flap may become palatal). It is then continued through the next
A B
C D
FIGURE 3-14. Retromolar area modified suture technique (see text).
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26 Basics
Terminal End Loop. On completion of suturing, Modification. When two flaps have been Alternative Procedure. This technique simulta-
the suture is tied off against the tooth as opposed reflected and after the first flap has been sutured neously slings together both the buccal and lin-
to the other flap. This is accomplished by leaving a (Figure 3-16A), it is often desirable to continue gual or palatal flaps.
loose loop of approximately 1 cm length of suture about the distal surface of the last tooth (Figure INDICATIONS.
material before the last embrasure. When the last 3-16B), repeating the procedure on the opposing 1. When flap position is not critical
papilla is sutured and the needle is returned flap (Figure 3-16C) and then tying off in a termi- 2. When buccal periosteal sutures are used for
through the embrasure, the terminal end loop is nal end loop (Figure 3-16D and E). buccal flap position and stabilization
used to tie the final knot (Figure 3-15F–I). 3. When maximum closure is desired (unrepo-
sition or Widman flaps or bone regeneration)
4
1 3
2
A B C
D E F
G H I
Technique Technique. The procedure is identical to that through the remaining loop of the suture, and the
After the initial buccal and lingual tie, the suture is previously described for the independent papil- suture is pulled tightly, thus locking it. This proce-
passed buccally about the neck of the tooth inter- lary sling suture (see Figure 3-15), except that dure is continued until the final suture is tied off at
dentally and through the lingual flap. It is then vertical or horizontal mattress sutures are substi- the terminal end (Figure 3-18).
again brought interdentally through the buccal tuted for the simple papillary sling. The tech-
papilla and back interdentally about the lingual nique is similar to that previously described for Suture Removal
surface of the tooth to the buccal papilla. Then it is the interrupted mattress sutures.
Sutures are used for wound stabilization and
brought about the lingual papilla and then the Locking. The continuous locking suture is indi- should be removed when sufficient tissue strength
buccal surface of the tooth. This alternating buc- cated primarily for long edentulous areas, has been achieved. This is usually between 5 and
cal-lingual suturing is continued until the suture is tuberosities, or retromolar areas. It has the advan- 10 days, and in most instances, these sutures are
tied off with a terminal end loop (Figure 3-17). tage of avoiding the multiple knots of interrupted removed in 7 days.
Vertical and Horizontal Mattress Suture. When sutures. If the suture is broken, however, it may
greater papillary control and stability and more completely untie. Materials
precise placement are required or to prevent flap Technique. The procedure is simple and repeti- 1. Scissors
movement, vertical or horizontal mattress sutures tive. A single interrupted suture is used to make the 2. Cotton pliers
are used. This is most often the case on the palate, initial tie. The needle is next inserted through the 3. Double-ended scaler
where additional tension is often required, or outer surface of the buccal flap and the underlying 4. Hydrogen peroxide
when the papillary tissue is thin and friable. surface of the lingual flap. The needle is then passed 5. Topical anesthetic
6. Cotton swabs
5
7 6
5 8
6
7
8
4
1
3
2
A B C
5
8 7 6
1 3
2
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