Abc of Burns: Kanwal Khan Lecturer ZCPT
Abc of Burns: Kanwal Khan Lecturer ZCPT
Abc of Burns: Kanwal Khan Lecturer ZCPT
Kanwal khan
Lecturer
ZCPT
WHAT ARE BURNS???
• Restore function—
• Maximize patient’s ability to perform pre-injury activities
• Restore feeling—
• Enable psychological and emotional recovery
CARE OF A MAJOR BURN INJURY
• Burn management can be divided up into seven phases—
1. Rescue
2. Resuscitate
3. Retrieve
4. Resurface
5. Rehabilitate
6. Reconstruct
7. Review
THE BODY’S RESPONSE TO BURN
• Local response
• Zone of coagulation—
• This occurs at the point of maximum damage.
• irreversible tissue loss due to coagulation of proteins.
• Zone of stasis—
• surrounding zone of decreased tissue perfusion.
• tissue in this zone is potentially salvageable.
• Additional insults—
• such as prolonged hypotension, infection, or oedema
• can convert into an area of complete tissue loss.
• Zone of hyperaemia—
• outermost zone
• tissue perfusion is increased.
• invariably recover unless there is severe sepsis or prolonged hypoperfusion.
THE BODY’S RESPONSE TO A BURN
• Systemic response
• once the burn reaches 30%
of total body surface area
• Electrical injuries
1. Electrocution
• Domestic electricity—
• The alternating nature of domestic current can interfere with the cardiac cycle, giving rise to arrhythmias.
MECHANISMS OF INJURY
• Chemical injuries
1. Industrial accidents
2. Household chemical products
• Acid or alkali
• deep, corrosive agent cause coagulative necrosis until completely removed.
• Non-accidental injury
1. Sexual abuse
2. Household abuse
ASSESSMENT OF
BURN AREA
• Wallace rule of nines
ASSESSMENT OF
BURN AREA
• Lund and Browder chart
FIRST AID
• Stop the burning process
1. Remove the source
• Analgesia
1. Cooling and covering
2. Opioids
3. NSAIDS
• B—Breathing
• Mechanical restriction of breathing
• Blast injury
• Smoke inhalation or Carboxyhaemoglobin
• C—Circulation
• Check pulse and circulation (Peripheral and central)??
• Intravenous access
• D—Neurological disability
• Responsiveness via GCS
• confused because of hypoxia or hypovolaemia.
• Superficial dermal—extends through the epidermis into the upper layers of the dermis
and is associated with blistering
• Deep dermal—extends through the epidermis into the deeper layers of the dermis but
not through the entire dermis
ASSESMENT OF BURN TYPE
ICU MANAGEMENT AND COMPLICATIONS
• TREATMENT
• Restricting fluids increases mortality
• If in doubt, intubate
• Give 100% oxygen until carbon monoxide toxicity excluded
• Ventilatory strategies to avoid lung injury (low volume or pressure)
• Aggressive airway toilet
• Early surgical debridement of wounds
• Early enteral feeding
ICU MANAGEMENT AND COMPLICATIONS
• Heart failure
• circulating myocardial depressant factor
• myocardial diastolic dysfunction
• myocardial oedema.
• TREATMENT
• Inotropic Agents
ICU MANAGEMENT AND COMPLICATIONS
• Kidney failure
• TREATMENT
• Early renal support (haemodialysis or haemodiafiltration)
ICU MANAGEMENT AND COMPLICATIONS
• Cerebral failure
• Hypoxic cerebral insults
• closed head injuries (DAI)
• TREATMENT
• Monitoring intracranial pressure
ICU MANAGEMENT AND COMPLICATIONS
• Nutrition
• Hypermetabolic Response
• TREATMENT
• Delayed healing
• Graft failure
• Inhalational injury
• Oedema management
• Compression—such as Coban oedema gloves
• Movement—rhythmic, pumping
• Elevation or positioning of limbs — for gravity assisted flow of oedema
• Maximization of lymphatic function
• Splinting does not control oedema but channel fluid to an immobile area.
REHABILITATION AFTER BURN INJURY
• Immobilisation
REHABILITATION AFTER BURN INJURY
• Scar management
• Hypertrophic scarring
• results from the build up of excess collagen fibres during wound healing
• the reorientation of those fibres in non-uniform patterns.
• Keloid scarring
• extends beyond the boundary of the initial injury.
• More common in people with pigmented skin than in white people.
• Management
• Pressure garments
• massage
• moisturising creams
• Therapeutic ultrasound