Developmental Disturbances of Teeth

Developmental Disturbances
of Teeth
Abnormalities in
Number
Eruption
Size
Shape
Structure
Abnormalities in Number of Teeth
Anodontia
Hyperdontia
Total
Streeter’s Syndrome
Partial Anodontia
Hereditary Anhydrotic Ectodermal Dysplasia
(Streeter’s Syndrome)
Partial Anodontia
Hereditary
Environmental
Local Factors
Trauma-
Infection-
Irradiation-
Systemic
Hormonal disturbances -
Germinal Measles -
Down’s syndrome-
Partial anodontia
true false
pseudo
Hyperdontia
Supplemental teeth
Supernumerary teeth
Pre-decideous
Post permanent
Extra 2
Supernumerary teeth
Mesiodense
Distomolar
Paramolar
Cleidocranial Dysplasia
Abnormalities in size of teeth
Macrodontia Microdontia
Generalized-1
localized-2
Abnormalities in Shape
Gemination Fusion
Partial division of single tooth germ Union of adjacent tooth
Upper and lower central incisors
partial or complete
hereitary
Trauma trauma, crowding
Number not affected number is decreased

Concrescence
Dilaceration
Taurodontism (bull like teeth)
Tooth is rectangular in shape

Enlarged pulp chamber which
lacks cervical constriction
Dens Invaginatus
)Dens in Dent(
Caused by invagination of all layers of enamel organ into dental

papilla during odontogenesis resulting in formation of an enamel
lined cavity which communicate with surface
Food accumulation, pulp infection and periapical disease
Radiographically: a tooth like structure appears within

the pulp of the involved tooth Dens in dent
Dens Evaginatus
Due to proliferation and evagination of enamel epithelium and
subjacent odontogenic mesenchme during tooth development
Appears as an extra cusp on occlosal surface between buccal and

lingual cusps of premolars. Unilaterally or bilaterally
Leon’s premolar
Extra cusp projecting lingually from

cingulum of maxillary and mandibular
permenant incisors and extends to the
.incisal edge
Resembling eagle’s talon
Talon cusp
Congenital Syphilis
Moon’s molar
Hutchinson's incisor
barrel shape, mesial and distal surfaces converge colossally
Central notch
Mesial and distal incisal angles are rounded off
A-Developmental Defects of Enamel
Hereditary
Acquired
Amelogenesis Imperfecta
Enamel Hypoplasia
Local Factors
Hereditary Hereditary
Enamel Enamel
Systemic Factors
Hypoplasia Hypocalcification
Hereditary Enamel Hypoplasia
Hereditary Enamel Hypocalcification

Acquired Enamel Hypoplasia
Local factors
Systemic Factors
)toxic damage to the ameloblasts(
Trauma Involve teeth formed during the first
)year after birth
Infection Nutritional deficiency-
Irradiation Vit def (A,D), calcium, phosphorous
Hormonal disturbances-
Infections-
Turner’s tooth
)scarlet fever, chicken pox, measels(
yellowish brown stain or Congenital syphilis-
pitting of enamel Ingestion of chemicals (fluoride,-
tetracyclin)
Or absence of enamel on
labial surface
Dental Fluorosis (Mottled Enamel)
Mild
Severe
Moderate
Tetracyclin pigmentation
Tetracyclin binds to calcium and precipitate along the

incremental lines of hard mineralized structures as
bone, dentine and enamel
The teeth are discolored and exhibit fluorescence under

ultraviolet light
Tetracyclin Pigmentation
B-Developmental Defects of Dentine
Hereditary
Acquired
Dentinogenesis Imperfecta
Dentinal Dysplasia
(Rootless tooth)
Type 1
Type 2 Hereditary brown opalescent dentine
Type 3 Shell, Pink or Brandy Wine tooth
both deciduous and permanent are affected-

crowns of teeth are shorter than normal with constricted neck giving-
bulbous appearance
enamel is normal but rapidly lost because it is un supported-
with healthy dentine, undermined enamel
short roots-
Radiographically
Partial or total obliteration of the pulp chambers and root
canals
Hereditary brown opalescent dentine
Dentinogenesis Imperfecta (Shell Tooth)
Enamel is normal
Peripheral layer of dentine is normal after that
formation of dentine stops thus there is wide pulp
.chamber surrounded by thin shell of dentine
Early pulp exposure
Roots are extremely short
Dentinogenesis Imperfecta (Shell Tooth)
Abnormalities in Eruption
Premature eruption
Submerged teeth
Delayed eruption
Embedded and impacted teeth
Hereditary
Hyperthyrodism Force space(obstruction)
Facial hemihypertrophy crowding
premature loss of dec
Systemic causes
rickets, hypothyrodism, mongolism
Local causes
retained dec, superneumerary, odontome

Developmental Disturbances of Teeth

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Developmental Disturbances

Upper and lower central incisors

Trauma trauma, crowding

Number not affected number is decreased

Tooth is rectangular in shape

Caused by invagination of all layers of enamel organ into dental

Food accumulation, pulp infection and periapical disease

Radiographically: a tooth like structure appears within

Appears as an extra cusp on occlosal surface between buccal and

Extra cusp projecting lingually from

Hereditary Enamel Hypocalcification

Tetracyclin binds to calcium and precipitate along the

The teeth are discolored and exhibit fluorescence under

both deciduous and permanent are affected-

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