Pages that link to "Q48464739"

The following pages link to Complement activation in chromosome 13 dementias. Similarities with Alzheimer's disease (Q48464739):

Displaying 27 items.

• Using animal models to determine the significance of complement activation in Alzheimer's disease (Q24796236) (← links)
• A Chronic Fatigue Syndrome - related proteome in human cerebrospinal fluid (Q25256334) (← links)
• Generation and initial characterization of FDD knock in mice (Q28750248) (← links)
• Genetics and molecular pathogenesis of sporadic and hereditary cerebral amyloid angiopathies (Q33747695) (← links)
• Aging and cerebrovascular dysfunction: contribution of hypertension, cerebral amyloid angiopathy, and immunotherapy (Q34219814) (← links)
• Genetic alterations of the BRI2 gene: familial British and Danish dementias (Q34513577) (← links)
• Severe ataxia with neuropathy in hereditary gelsolin amyloidosis: a case report (Q34622028) (← links)
• Memory deficits due to familial British dementia BRI2 mutation are caused by loss of BRI2 function rather than amyloidosis (Q34667384) (← links)
• Amyloidosis associated with cerebral amyloid angiopathy: cell signaling pathways elicited in cerebral endothelial cells. (Q35741865) (← links)
• Neurodegeneration caused by proteins with an aberrant carboxyl-terminus (Q35871644) (← links)
• Islet amyloid polypeptide triggers limited complement activation and binds complement inhibitor C4b-binding protein, which enhances fibril formation (Q35879624) (← links)
• Structural and functional anatomy of the globular domain of complement protein C1q. (Q35896873) (← links)
• Memory deficits of British dementia knock-in mice are prevented by Aβ-precursor protein haploinsufficiency. (Q35917231) (← links)
• Proteomic characterization of a mouse model of familial Danish dementia (Q35955282) (← links)
• Interaction of ApoE3 and ApoE4 isoforms with an ITM2b/BRI2 mutation linked to the Alzheimer disease-like Danish dementia: Effects on learning and memory (Q36988218) (← links)
• Cerebral amyloid angiopathy and parenchymal amyloid deposition in transgenic mice expressing the Danish mutant form of human BRI2. (Q37019428) (← links)
• Expression of BRI2 mRNA and protein in normal human brain and familial British dementia: its relevance to the pathogenesis of disease (Q37481580) (← links)
• Clusterin and complement activation in exfoliation glaucoma (Q37714090) (← links)
• Complement in the brain. (Q37872442) (← links)
• Papaya glutamine cyclotransferase shows a singular five-fold beta-propeller architecture that suggests a novel reaction mechanism. (Q41451534) (← links)
• Gene expression profiling of aging in multiple mouse strains: identification of aging biomarkers and impact of dietary antioxidants (Q41866466) (← links)
• CEREBRAL AMYLOID ANGIOPATHY AND ALZHEIMER'S DISEASE. (Q42396970) (← links)
• Fibrillation of the major curli subunit CsgA under a wide range of conditions implies a robust design of aggregation (Q43072878) (← links)
• Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease (Q43249892) (← links)
• Molecular chaperons, amyloid and preamyloid lesions in the BRI2 gene-related dementias: a morphological study (Q48421288) (← links)
• pH-Dependent Amyloid and Protofibril Formation by the ABri Peptide of Familial British Dementia (Q54126071) (← links)
• Withdrawn: Severe ataxia with neuropathy in hereditary gelsolin amyloidosis (Q58300506) (← links)