Location via proxy:   [ UP ]  
[Report a bug]   [Manage cookies]                
Scribd Logo
Upload

Welcome to Scribd!

  • 35 views

    Pa Tho Physiology

    Uploaded by

    Arnel Luces Beato
    The document discusses modifiable and non-modifiable risk factors for multinodular non-toxic goiter. It outlines the pathophysiology which involves iodine deficiency, poor socioeconomic status, and heredity leading to stimulation of the thyroid-stimulating hormone (TSH) receptor and formation of multiple thyroid nodules. It then describes treatment with propylthiouracil (PTU) to normalize thyroid hormone levels and maintenance therapy for two months.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd

    Pa Tho Physiology

    Uploaded by

    Arnel Luces Beato
    35 views2 pages
    The document discusses modifiable and non-modifiable risk factors for multinodular non-toxic goiter. It outlines the pathophysiology which involves iodine deficiency, poor socioeconomic status, and heredity leading to stimulation of the thyroid-stimulating hormone (TSH) receptor and formation of multiple thyroid nodules. It then describes treatment with propylthiouracil (PTU) to normalize thyroid hormone levels and maintenance therapy for two months.

    Copyright

    © Attribution Non-Commercial (BY-NC)

    Available Formats

    DOC, PDF, TXT or read online from Scribd

    Did you find this document useful?

    Is this content inappropriate?

    The document discusses modifiable and non-modifiable risk factors for multinodular non-toxic goiter. It outlines the pathophysiology which involves iodine deficiency, poor socioeconomic status, and heredity leading to stimulation of the thyroid-stimulating hormone (TSH) receptor and formation of multiple thyroid nodules. It then describes treatment with propylthiouracil (PTU) to normalize thyroid hormone levels and maintenance therapy for two months.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Download as doc, pdf, or txt
    35 views2 pages

    Pa Tho Physiology

    Uploaded by

    Arnel Luces Beato
    The document discusses modifiable and non-modifiable risk factors for multinodular non-toxic goiter. It outlines the pathophysiology which involves iodine deficiency, poor socioeconomic status, and heredity leading to stimulation of the thyroid-stimulating hormone (TSH) receptor and formation of multiple thyroid nodules. It then describes treatment with propylthiouracil (PTU) to normalize thyroid hormone levels and maintenance therapy for two months.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Download as doc, pdf, or txt
    of 2

    Modifiable factors:

    Non-modifiable
    Iodine
    factors:
    deficiency Mild
    female PATHOPHYSIOLOGY
    Poor Hoarse
    age
    socioeconomic ness of
    heredity
    T3 and T4 status ice

    Stimulation of laryn lymphocytes: 0.55,


    Throug
    TSH gitis Segmenter: 0.72
    h TSH
    receptor Induction of thyroid cell
    s hyperplasia Formation of dyspha
    multinodular goiter gia
    Increased replication
    predisposes to a risk of UTZ shows
    mutation in the TSH Palpable
    three cystic
    receptors nodule on
    nodules
    the right
    (0.4cm) in
    side of the
    Mutated TSH receptor is the right
    neck
    constitutively active lobe

    produced excess T3/T4 For biopsy


    (not applicable
    to indigent
    patient)
    HYPERTHYROIDISM

    PTU 50mg TID


    maintenance for 2 months TSH Test
    shows
    normal T3: 3.47, T4:
    Normalized thyroid 1.21 and
    hormones level level of
    T3,T4 and TSH: 1.89
    TSH
    Multinodular Non-toxic Total
    Goiter thyroidect
    omy

    You might also like