FrmPatientViewAllReport - 2023-04-06T211159.013 PDF
FrmPatientViewAllReport - 2023-04-06T211159.013 PDF
FrmPatientViewAllReport - 2023-04-06T211159.013 PDF
HAEMATOLOGY
HEALTHKIND ACTIVE
Complete Blood Count (CBC)
Haemoglobin (Hb) 16.0 13 - 17 gm/dL
Sample: Whole Blood EDTA
Method: Photometric measurement
MCH 30.2 27 - 32 pg
Sample: Whole Blood EDTA
Method: Calculated
Neutrophils 55 40 - 80 %
Sample: Whole Blood EDTA
Method: VCS Technology & Microscopy
Lymphocytes 37 20 - 40 %
Sample: Whole Blood EDTA
Method: VCS Technology & Microscopy
Eosinophils 05 1-6 %
Sample: Whole Blood EDTA
Method: VCS Technology & Microscopy
Monocytes 02 2 - 10 %
Sample: Whole Blood EDTA
Method: VCS Technology & Microscopy
Basophils 01 0-2 %
Sample: Whole Blood EDTA
Method: VCS Technology & Microscopy
Lipid Profile
Total Cholesterol 171 No risk : < 200 mg/dL
Sample: Serum Moderate risk : 200–239
Method: Spectrophotometery High risk : =240
Clinical Significance :
CBC comprises of estimation of the cellular componenets of blood including RBCs, WBCs and Platelets. Mean corpuscular volume (MCV) is a
measure of the size of the average RBC, MCH is a measure of the hemoglobin cointent of the average RBC and MCHC is the hemoglobin
concentration per RBC. The red cell distribution width (RDW) is a measure of the degree of variation in RBC size (anisocytosis) and is helpful in
distinguishing between some anemias. CBC examination is used as a screening tool to confirm a hematologic disorder, to establish or rule out a
diagnosis, to detect an unsuspected hematologic disorder, or to monitor effects of radiation or chemotherapy. Abnormal results may be due to a
primary disorder of the cell-producing organs or an underlying disease. Results should be interpreted in conjunction with the patient's clinical picture
and appropriate additional testing performed.
Clinical Significance :
Hemoglobin A1c (HbA1c) level reflects the mean glucose concentration over the previous period (approximately 8-12 weeks) and provides a much
better indication of long-term glycemic control than blood and urinary glucose determinations. American Diabetes Association (ADA) include the use
of HbA1c to diagnose diabetes, using a cutpoint of 6.5%. The ADA recommends measurement of HbA1c 3-4 times per year for type 1 and poorly
controlled type 2 diabetic patients, and 2 times per year for well-controlled type 2 diabetic patients) to assess whether a patient's metabolic control
has remained continuously within the target range. Falsely low HbA1c results may be seen in conditions that shorten erythrocyte life span. and may
not reflect glycemic control in these cases accurately.
Lipid Profile
Proposed LDL-C goals in very high risk and extreme risk group patients by the Lipid Association of India.
The LDL-C goal of ≤30 mg/dl must be pursued after detailed risk-benefit discussion between physician and patient.
Clinical judgment to be used in decision making if the patient has disease/risk factors not covered in the table, eg. peripheral arterial disease or
cerebrovascular disease.
∗Major ASCVD risk factors: 1. Age- male ≥45 years, female ≥55 years, 2. Family h/o premature CAD- male <55 years, female <65 years, 3.
Smoking/tobacco use, 4. Systemic hypertension, 5.Low HDL (males <40 mg/dl and females <50 mg/dl).
#Moderate non-conventional risk factors: 1. Coronary calcium score 100–299 HU, 2. Increased carotid intima-media thickness, 3. Lp(a) ≥20–49
Uric Acid
Clinical Significance :
Uric acid is the final product of purine metabolism. Serum uric acid levels are raised in case of increased purine synthesis, inherited metabolic disorder,
excess dietary purine intake, increased nucleic acid turnover, malignancy and cytotoxic drugs. Decreased levels are seen in chronic renal failure, severe
hepatocellular disease with reduced purine synthesis, defective renal tubular reabsorption, overtreatment of hyperuricemia with allopurinol, as well as
some cancer therapies.
Total T3 (Triiodothyronine)
Clinical Significance :
Thyroid hormones, T3 and T4, which are secreted by the thyroid gland, regulate a number of developmental, metabolic, and neural activities throughout
the body. The thyroid gland synthesizes 2 hormones - T3 and T4. T3 production in the thyroid gland constitutes approximately 20% of the total
circulating T3, 80% being produced by peripheral conversion from T4. T3 is more potent biologically. Total T3 comprises of Free T3 and bound T3.
Bound T3 remains bound to carrier proteins like thyroid-binding globulin, prealbumin, and albumin). Only the free forms are metabolically active. In
hyperthyroidism, both T4 and T3 levels are usually elevated, but in some rare cases, only T3 elevation is also seen. In hypothyroidism T4 and T3
levels are both low. T3 levels are frequently low in sick or hospitalized euthyroid patients.
Total T4 (Thyroxine)
Clinical Significance :
Total T4 is synthesized in the thyroid gland. About 0.05% of circulating T4 is in the free or biologically active form. The remainder is bound to
thyroxine-binding globulin (TBG), prealbumin, and albumin. High levels of T4 (and FT4) causes hyperthroidism and low levels lead to
hypothyroidism.
Clinical Significance :
TSH levels are elevated in primary hyporthyroidism and low in primary hyperthyroidism. Evaluation of TSH is useful in the differential diagnosis of
primary from secondary and tertiary hypothyroidism. In primary hypothyroidism, TSH levels are elevated, while in secondary and tertiary
hypothyroidism, TSH levels are low or normal. High TSH level in the presence of normal FT4 is called subclinical hypothyroidism and low TSH with
normal FT4 is called subclinical hyperthyroidism. Sick, hospitalized patients may have falsely low or transiently elevated TSH. Significant diurnal
variation is also seen in TSH levels.
** End of Report**