Head Trauma: Kelompok 2: Andry Wongso Rhiza M.I.S Terri S. S Priscilla
Head Trauma: Kelompok 2: Andry Wongso Rhiza M.I.S Terri S. S Priscilla
Head Trauma: Kelompok 2: Andry Wongso Rhiza M.I.S Terri S. S Priscilla
Kelompok 2:
Andry Wongso
Rhiza M.I.S
Terri S. S
Priscilla
Pembimbing:
Prof. Dr. dr. M. Z. Arifin, SpBS (K)
Intracranial Pressure
Pressure / Volume Curve
ICP
Herniation
10 point of decompensation
volume of mass
PressureICP
Blunt:
automobile collision, fall & assault
Penetrating:
gunshot wounds, other penetrating
injuries
- Severity
Mild:
GCS score 14 ~ 15
Moderate:
Severe:
GCS score 3 ~ 8
Skull fractures
Vault:
linear / stellate,
depressed / nondepressed,
open / close
Basilar (diagnosed by CT bone window):
raccoon eyes, Battles signs
(retroauricular ecchymosis),
CSF leakage and 7th nerve palsy
Intracranial Lesions
Focal lesions:
Epidural hematoma:
most due to tearing of the middle meningeal artery
prognosis is usually excellent ( underlying brain
injury is limited )
CT: biconvex or lenticular in shape
Pitfalls: classical lucid interval and talk and die
Intracranial Lesions
Focal lesions
Subdural hematoma:
brain damage much more & prognosis is much
worse than EDH
tearing of a bridging vein
Focal lesions
Contusions and intracerebral hematomas:
most occur in the frontal & temporal
lobes
always seen in association with SDH
Intracranial Lesions
Diffuse injuries
Mild concussion: temporary neurologic dysfunction,
confusion & disorientation without or with amnesia
Classic cerebral concussion:
1.Transient & reversible loss of consciousness, returns
to full consciousness by 6 hrs.
2.No sequelae other than amnesia for the events
3.post-concussion syndrome: memory difficulties,
dizziness, nausea, anosmia & depression
Intracranial Lesions
Diffuse injuries:
Diffuse axonal injury ( DAI )
1.prolonged postraumatic coma that
is not due to a mass lesion or
ischemic insults
2.usually having decortication or
decerebation posture
3.autonomic dysfunction:
hypertension, hyperhidrosis &
hyperpyrexia
Vital Signs
Identifies neurologic & systemic status
Presume hypotension due to hypovolemia,
not head injury
Minineurologic Exam
Purpose
Determine severity of brain injury
Detect deterioration
Categories injuries
Minineurologic Exam
Pupils
Size
Equality
Briskness of response
Anormal: >1 mm difference in size
Extremity Movement
Equality
Pain response
Lateralized weakness - mass lesion
Diagnostic Procedure
CT:
be obtained in all head -injury patients ( ideally ), especially there
is a history of more than a momentary loss of consciousness,
amnesia or severe headaches
C-Spine
Alcohol level & urine toxic screen
Skull X-ray:
penetrating head injury or when CT scan is not immediately
available
Intravenous Fluid:
1. Keep euvolemic status, dehydration is
more
harmful ( vital signs stable )
2. Not to use hypotonic or glucose-containing fluids
Hyperventilation:
1. Keep PaCO2 at 25~30 mmHg when the
presence of raised
ICP
2. PaCO2 < 25 mmHg is avoided ( vasoconstriction ==> CBF
)
Steroid :
Not demonstrated any beneficial effect
Anticonvulsants
High incidence of Late epilepsy:
1. Early seizure occurring within the first week
2. An intracranial hematoma
3. Depressed skull fracture
phenytoin reduce the incidence of seizure in the first week of injury
but not thereafter
TERIMA KASIH
22