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B.SC Nursing Medical Surgical Nursing - I Unit: Iv - Nursing Management of Patients With Disorders of Digestive System Portal Hypertension

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B.

Sc NURSING
MEDICAL SURGICAL NURSING – I
UNIT: IV -NURSING MANAGEMENT OF
PATIENTS WITH DISORDERS OF
DIGESTIVE SYSTEM
PORTAL HYPERTENSION

MR.V.POOVARAGAVAN.RN
PROFESSOR
OBJECTIVES

At the end of the class the students are able to


- Define portal hypertension

- Describe the risk factor, etiology of portal


hypertension
- Explain the pathophysiology and clinical
manifestation of portal hypertension
OBJECTIVES

At the end of the class the students are able to


- Discuss the diagnostic evaluation and
complication of portal hypertension
- enumerate medical, surgical and nursing
management of patient with portal hypertension
INTRODUCTION

Portal Veins coming from the stomach,


intestine, spleen , and  pancreas  merge into the
portal vein, which then branches into smaller
vessels and travels through the  liver. If the
vessels in the  liver are blocked due to liver
damage, blood cannot flow properly through the
liver.
ANATOMY AND PHYSIOLOGY

Portal veins coming from the stomach,


intestine, spleen , and  pancreas  merge into the
portal vein, which then branches into smaller
vessels and travels through the  liver. If the
vessels in the  liver are blocked due to liver
damage, blood cannot flow properly through the
liver.
ANATOMY AND PHYSIOLOGY

As a result, high pressure in the portal


system develops. This increased pressure in the
portal vein may lead to the development of large,
swollen veins (varices) within the esophagus
stomach, rectum, or umbilical area (belly button).
Varices can rupture and bleed, resulting in
potentially life-threatening complications.
DEFINITION
Portal hypertension is elevated pressure in the portal
vein associated with increased resistance to blood
flow through the portal venous system.

Portal hypertension is defined as the elevation of the


hepatic venous pressure gradient to > 5 mmhg.

Clinically significant portal hypertension is present


when gradient exceeds 10 mmHg. Risk of variceal
bleeding increases beyond a gradient of 12 mmHg.
CLASSIFICATION AND CAUSES

Prehepatic

Portal vein thrombosis

Splenic vein thrombosis

Massive spleenomegaly
CLASSIFICATION AND CAUSES

Hepatic

1. Presinusoidal: schistosomiasis

congenital hepatic fibrosis

2. Sinusoidal: cirrhosis of liver alcoholic

hepatitis

3. Postsinusoidal: hepatic sinusoidal

obstruction (veno-occlusive syndrome)


CLASSIFICATION AND CAUSES

Post hepatic

-Budd-Chiari Syndrome

-Inferior vena cava obstruction

-cardiac causes:

Restrictive cardiomyopathy

Constrictive pericarditis

Severe congestive cardiac failure


PATHOPHYSIOLOGY

Due to causes

Obstruction of portal venous flow

Portal vascular resistance is increased in liver cell injury

Stellate cell activation into myofibroblast & smooth muscle protein

alpha-actin Endothelin Contraction of activated cells

Abnormal blood flow pattern causing increased resistance

fibrogenesis Increased resistance leads to portal hypertension


SIGNS AND SYMPTOMS
• Ascites

• Shifting dullness or fluid wave on abdominal


percussion
• Dilated abdominal vessels radiating from the
umbilicus (e.g. caput medussae)
• Enlarged, palpable spleen

• Bruits detected over the upper abdominal area


because of esophageal and gastric varicosities.
DIAGNOSTIC EVALUATION

 HISTORY COLLECTION

 PHYSICAL EXAMINATION

 BLOOD TESTS.

 CHEST X RAY

 ANGIOGRAM

 ULTRASOUND

 ENDOSCOPY
MEDICAL MANAGEMENT

• Beta-blockers and nitroglycerin may be
appropriate to decrease pressure within the
portal system.
• Lactulose may be prescribed as a treatment for
hepatic encephalopathy.
• propranolol and isosorbide, may help lower the
pressure in the portal vein
NON MEDICAL MANAGEMENT

Dietary restrictions include limiting salt to


prevent further ascites fluid accumulation.
Protein may also be restricted, since increased
protein load can overwhelm the liver's ability to
synthesize it and may lead to hepatic
encephalopathy.
NON MEDICAL MANAGEMENT

Distal splenorenal shunt (DSRS) was

designed to reroute blood only from the veins

coming from the esophagus and stomach while

preserving the blood flow through the portal vein.

The splenic vein was joined to the left

kidney vein thereby selectively decompressing

the esophageal and gastric varices.


DISTAL SPLENORENAL SHUNT (DSRS)
MEDICAL MANAGEMENT

Sclerotherapy is the techniques of

injecting sclerosing drugs into the varices,

causing a narrowing of the swollen veins thus

preventing bleeding and reducing swelling. This

procedure is done endoscopically.


SURGICAL MANAGEMENT
Transjugular intrahepatic portosystemic shunt
(TIPS), a catheter is introduced by a radiologist into
the jugular vein and advanced to the hepatic vein.

The catheter is threaded into a large branch of the


portal vein, and a stent is placed connecting the portal
vein (bringing blood to the liver from the digestive
tract) with the hepatic vein (returning blood from the
liver to the heart).
TRANSJUGULAR INTRAHEPATIC
PORTOSYSTEMIC SHUNT (TIPS)
NURSING DIAGNOSIS

• Risk for infection

• Activity intolerance

• Risk for imbalanced fluid volume


NURSING MANAGEMENT
• Administer medications, which may include diuretics.

• Assist the health care provider with paracentesis,


which removes the fluid (e.g. ascites) from the
peritoneal cavity; the volume usually is limited to 2 to
3L of fluid, but it may be more. Observe the client
closely for signs and symptoms of vascular collapse.

• Measure and record abdominal girth and body


weight daily, assess for abdominal fluid wave.
NURSING MANAGEMENT

• Promote measures to prevent or reduce edema.

• Encourage the client to elevate the lower

extremities and wear support hose to prevent

lower-extremity edema.
NURSING MANAGEMENT

• Administer salt-poor albumin, which temporarily

elevates the serum albumin level. This increases

serum osmotic pressure, helping to reduce edema

by causing ascetic fluid to be drawn back into the

bloodstream and eliminated by the kidneys.


BIBLIOGRAPHY

 Brunner and suddarth’s (2004) “Text book of


medical surgical nursing”, 10th edition, published by
Lippincott Williams and Wilkins page no. 2079-2087.
 Lewis (2002), “Medical surgical nursing”, 6th edition,
published by Mosby page no 1635-1651.
 Black .J.M. & Hawks .J.H, (2004), “Medical Surgical
Nursing” 7th edition, New Delhi: Elsevier publication,

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