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    Neocerebellar Syndrome

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    goldengoal19079
    This document discusses the manifestations of neocerebellar syndrome. It outlines several symptoms: 1) hypotonia and muscle weakness, 2) motor ataxia characterized by dysmetria, dysarthria, disturbed gait, dysdiadokokinesia, decomposition of movement, rebound phenomenon, and kinetic tremor, 3) cerebellar nystagmus, which is involuntary eye movement. It provides definitions and descriptions of each symptom, explaining they result from failure of the cerebellum's role in damping, timing, predictive and smooth progression of movement. Tests like finger-to-nose are used to examine dysmetria, while heel-knee assesses decomposition of movement.

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    Neocerebellar Syndrome

    Uploaded by

    goldengoal19079
    25 views14 pages
    This document discusses the manifestations of neocerebellar syndrome. It outlines several symptoms: 1) hypotonia and muscle weakness, 2) motor ataxia characterized by dysmetria, dysarthria, disturbed gait, dysdiadokokinesia, decomposition of movement, rebound phenomenon, and kinetic tremor, 3) cerebellar nystagmus, which is involuntary eye movement. It provides definitions and descriptions of each symptom, explaining they result from failure of the cerebellum's role in damping, timing, predictive and smooth progression of movement. Tests like finger-to-nose are used to examine dysmetria, while heel-knee assesses decomposition of movement.

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    This document discusses the manifestations of neocerebellar syndrome. It outlines several symptoms: 1) hypotonia and muscle weakness, 2) motor ataxia characterized by dysmetria, dysarthria, disturbed gait, dysdiadokokinesia, decomposition of movement, rebound phenomenon, and kinetic tremor, 3) cerebellar nystagmus, which is involuntary eye movement. It provides definitions and descriptions of each symptom, explaining they result from failure of the cerebellum's role in damping, timing, predictive and smooth progression of movement. Tests like finger-to-nose are used to examine dysmetria, while heel-knee assesses decomposition of movement.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Download as pptx, pdf, or txt
    25 views14 pages

    Neocerebellar Syndrome

    Uploaded by

    goldengoal19079
    This document discusses the manifestations of neocerebellar syndrome. It outlines several symptoms: 1) hypotonia and muscle weakness, 2) motor ataxia characterized by dysmetria, dysarthria, disturbed gait, dysdiadokokinesia, decomposition of movement, rebound phenomenon, and kinetic tremor, 3) cerebellar nystagmus, which is involuntary eye movement. It provides definitions and descriptions of each symptom, explaining they result from failure of the cerebellum's role in damping, timing, predictive and smooth progression of movement. Tests like finger-to-nose are used to examine dysmetria, while heel-knee assesses decomposition of movement.

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    )-( LECTURE NO

    Neuro-Physiology
    Kamal Mohammed
    Lecturer Of Physiology

    Neocerebellar
    syndrome

    Faculty Of Medicine Head Dept.Of Physiology


    Manifestations
    1. Hypotonia:
    • with subsequent pendular knee jerk.

    • there is muscle weakness because there is


    difficulty in maintenance of muscle
    contraction.
    Motor (cerebellar) ataxia
    2. Motor (cerebellar) ataxia is manifested by:
    1-Dysmetria:
    • Definition: inability to adjust the movements
    to a certain distance.
    • Types:
    a. Hypermetria: overshoot of movement.
    b. Hypometria: stop short before its aim.
    • Cause: failure of damping and timing
    functions of the cerebellum.
    • Examination: by finger to nose test.
    2-Dysarthria:
    • Definition: difficulty in producing clear
    speech, due to failure of smooth progression
    and prediction of movements in muscles of
    larynx, mouth, and respiratory muscles.
    • Characters: the volume of sound is not
    controlled; it is either loud or weak speech
    with tendency to hesitate at the start of word
    (as Aaa..lll.eee.xxx..aanddria).
    3-Disturbance in gait:
    • the patient walks in a zigzag manner and tend
    to fall toward the affected side.
    3-Dysdiadokokinesia (adiadokokinasia
    ):
    • Definition: it is the inability to perform
    rapidly alternate opposite successive
    movement like supination and pronation.
    • Cause: failure of predictive and smooth
    progression function of the cerebellum.
    4-Decomposition of movement:
    • Definition: the patient has difficulty in
    performing simultaneous movements at more
    than one joint.
    • Character: patients dissect the movements
    and carry them one joint at a time.
    • Examination: by heel – knee test
    5- Rebound phenomenon:
    • Definition: an overshooting of a limb when a
    resistance to its movement is removed.
    • Normally: flexion of the forearm against
    resistance is checked when the resistance is broken
    off.
    • In cerebellar disease: the patient cannot stop
    movement of the limb and the forearm flies
    backward.
    • Cause: inability to stop the movement.
    6-Kinetic (intention) tremor:
    • Definition: when a person performs a
    voluntary act, the movements oscillate back
    and forth at the intended point, because
    overshooting of the movement at the intended
    point cause the motor cortex to correct the
    overshooting by an opposite movement, which
    again overshoot to the other side.
    • Characters:
    i. It continues until the movement finally settles
    to the intend mark.
    ii. It appears when the patient performs
    voluntary action.
    iii. It is absent at rest.
    7-Cerebellar nystagmus:
    • Involuntary, rapid oscillation of the eyeballs in
    a horizontal, vertical, or rotary direction, with
    the fast component maximal toward the side of
    the cerebellar lesion.
    • secondary to tremors in extraocular muscles.
    Testing of coordination of voluntary
    movements
    1-Eyes open or closed:
    I. Cerebellar ataxia is not improved by visual
    orientation.
    II. Sensory ataxia (from dorsal column disease)
    is worsened with closed eyes.
    Thanks

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