Synaptic changes in Alzheimer's disease: increased amyloid-beta and gliosis in surviving terminals is accompanied by decreased PSD-95 fluorescence (Q35103366)

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English	Synaptic changes in Alzheimer's disease: increased amyloid-beta and gliosis in surviving terminals is accompanied by decreased PSD-95 fluorescence	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

6 August 2017

Synaptic changes in Alzheimer's disease: increased amyloid-beta and gliosis in surviving terminals is accompanied by decreased PSD-95 fluorescence (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

Alzheimer's disease

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author name string

Karen Hoppens Gylys

1

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Europe PubMed Central

PubMed publication ID

6 August 2017

Jeffrey A Fein

2

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Europe PubMed Central

PubMed publication ID

6 August 2017

Fusheng Yang

3

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Europe PubMed Central

PubMed publication ID

6 August 2017

Dorothy J Wiley

4

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Europe PubMed Central

PubMed publication ID

6 August 2017

Carol A Miller

5

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Europe PubMed Central

PubMed publication ID

6 August 2017

Gregory M Cole

6

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6 August 2017

language of work or name

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publication date

1 November 2004

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Europe PubMed Central

PubMed publication ID

6 August 2017

language of work

The American Journal of Pathology

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Europe PubMed Central

PubMed publication ID

6 August 2017

165

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Europe PubMed Central

PubMed publication ID

6 August 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

1809-1817

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

Docosahexaenoic acid protects from dendritic pathology in an Alzheimer's disease mouse model

1 reference

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Amyloid beta peptide load is correlated with increased beta-secretase activity in sporadic Alzheimer's disease patients

1 reference

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9 July 2018

Beta-secretase activity increases with aging in human, monkey, and mouse brain.

1 reference

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9 July 2018

New roles for astrocytes: regulation of CNS synaptogenesis.

1 reference

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Anatomical aspects of glia-synapse interaction: the perisynaptic glial sheath consists of a specialized astrocyte compartment

1 reference

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Intraneuronal Alzheimer abeta42 accumulates in multivesicular bodies and is associated with synaptic pathology

1 reference

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9 July 2018

Alzheimer's disease is a synaptic failure

1 reference

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Do SNARE proteins confer specificity for vesicle fusion?

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The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

1 reference

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Drebrin, a dendritic spine protein, is manifold decreased in brains of patients with Alzheimer's disease and Down syndrome

1 reference

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Caspases, apoptosis, and Alzheimer disease: causation, correlation, and confusion

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Clustering and anchoring mechanisms of molecular constituents of postsynaptic scaffolds in dendritic spines

1 reference

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9 July 2018

Targeting small Abeta oligomers: the solution to an Alzheimer's disease conundrum?

1 reference

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Age-dependent changes in brain, CSF, and plasma amyloid (beta) protein in the Tg2576 transgenic mouse model of Alzheimer's disease.

1 reference

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Control of synapse number by glia

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Staging of cytoskeletal and beta-amyloid changes in human isocortex reveals biphasic synaptic protein response during progression of Alzheimer's disease

1 reference

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Ibuprofen suppresses plaque pathology and inflammation in a mouse model for Alzheimer's disease.

1 reference

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9 July 2018

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation

1 reference

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9 July 2018

Intraneuronal Abeta42 accumulation in human brain.

1 reference

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9 July 2018

"Apoptotic" biochemical cascades in synaptic compartments: roles in adaptive plasticity and neurodegenerative disorders

1 reference

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9 July 2018

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

1 reference

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Beta-amyloid induces local neurite degeneration in cultured hippocampal neurons: evidence for neuritic apoptosis

1 reference

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1 reference

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Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

1 reference

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Antibody to caspase-cleaved actin detects apoptosis in differentiated neuroblastoma and plaque-associated neurons and microglia in Alzheimer's disease

1 reference

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Synaptic efficacy enhanced by glial cells in vitro.

1 reference

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Disappearance of actin-binding protein, drebrin, from hippocampal synapses in Alzheimer's disease

1 reference

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Is the "preamyloid" of diffuse plaques in Alzheimer's disease really nonfibrillar?

1 reference

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The glutamatergic nerve terminal

1 reference

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The synaptic vesicle cycle: a cascade of protein-protein interactions

1 reference

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Domain interaction between NMDA receptor subunits and the postsynaptic density protein PSD-95

1 reference

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Synapse loss in frontal cortex biopsies in Alzheimer's disease: correlation with cognitive severity

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Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment

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Dimeric amyloid beta protein rapidly accumulates in lipid rafts followed by apolipoprotein E and phosphorylated tau accumulation in the Tg2576 mouse model of Alzheimer's disease.

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12 December 2020

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10.1016/S0002-9440(10)63436-0

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Europe PubMed Central

PubMed publication ID

6 August 2017

PMC publication ID

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Europe PubMed Central

PubMed publication ID

6 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

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