Pages that link to "Q46768341"
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The following pages link to Beta-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses (Q46768341):
Displaying 50 items.
- DHA improves cognition and prevents dysfunction of entorhinal cortex neurons in 3xTg-AD mice (Q21135531) (← links)
- Synapse Formation and Function Is Modulated by the Amyloid Precursor Protein (Q22241520) (← links)
- Progressive accumulation of amyloid-beta oligomers in Alzheimer's disease and in amyloid precursor protein transgenic mice is accompanied by selective alterations in synaptic scaffold proteins (Q24329069) (← links)
- Synaptic AMPA receptor plasticity and behavior. (Q24607574) (← links)
- Synthetic amyloid-beta oligomers impair long-term memory independently of cellular prion protein (Q24630045) (← links)
- Protein phosphatases and Alzheimer's disease (Q24632495) (← links)
- Relationships between expression of apolipoprotein E and beta-amyloid precursor protein are altered in proximity to Alzheimer beta-amyloid plaques: potential explanations from cell culture studies (Q24633831) (← links)
- Days to criterion as an indicator of toxicity associated with human Alzheimer amyloid-beta oligomers (Q24633883) (← links)
- The Emergence of NMDA Receptor Metabotropic Function: Insights from Imaging (Q26741815) (← links)
- Amyloid-β-Induced Dysregulation of AMPA Receptor Trafficking (Q26748723) (← links)
- Pathogenesis of synaptic degeneration in Alzheimer's disease and Lewy body disease (Q26853610) (← links)
- Striatal-enriched protein tyrosine phosphatase in Alzheimer's disease. (Q27687325) (← links)
- Trafficking and proteolytic processing of APP (Q28265871) (← links)
- Cargo trafficking in Alzheimer’s disease: the possible role of retromer (Q28268822) (← links)
- Disassembly of shank and homer synaptic clusters is driven by soluble beta-amyloid(1-40) through divergent NMDAR-dependent signalling pathways (Q28575781) (← links)
- Synaptic protein α1-takusan mitigates amyloid-β-induced synaptic loss via interaction with tau and postsynaptic density-95 at postsynaptic sites (Q28577234) (← links)
- Impaired balance of mitochondrial fission and fusion in Alzheimer's disease (Q28854335) (← links)
- Overrepresentation of glutamate signaling in Alzheimer's disease: network-based pathway enrichment using meta-analysis of genome-wide association studies. (Q29417035) (← links)
- AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine loss (Q29614850) (← links)
- Inhibition of calcineurin-mediated endocytosis and alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors prevents amyloid beta oligomer-induced synaptic disruption (Q30175697) (← links)
- Reversal of fragile X phenotypes by manipulation of AβPP/Aβ levels in Fmr1KO mice (Q30473360) (← links)
- Amyloid-beta and mitochondria in aging and Alzheimer's disease: implications for synaptic damage and cognitive decline (Q30476347) (← links)
- Synaptic activity reduces intraneuronal Abeta, promotes APP transport to synapses, and protects against Abeta-related synaptic alterations (Q30491040) (← links)
- Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice. (Q30497246) (← links)
- Phospholipase d2 ablation ameliorates Alzheimer's disease-linked synaptic dysfunction and cognitive deficits (Q30497694) (← links)
- Amyloid beta from axons and dendrites reduces local spine number and plasticity. (Q30511922) (← links)
- Alzheimer amyloid beta inhibition of Eg5/kinesin 5 reduces neurotrophin and/or transmitter receptor function (Q30583064) (← links)
- Lowering beta-amyloid levels rescues learning and memory in a Down syndrome mouse model (Q33598454) (← links)
- c-Jun N-terminal kinase has a key role in Alzheimer disease synaptic dysfunction in vivo. (Q33695108) (← links)
- Dysregulation of the mTOR pathway mediates impairment of synaptic plasticity in a mouse model of Alzheimer's disease (Q33700331) (← links)
- Behavioral alterations are associated with vitamin B12 deficiency in the transcobalamin receptor/CD320 KO mouse (Q33703586) (← links)
- GluA1 subunit ubiquitination mediates amyloid-β-induced loss of surface α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors (Q33705492) (← links)
- Genetic modulation of soluble Aβ rescues cognitive and synaptic impairment in a mouse model of Alzheimer's disease (Q33707872) (← links)
- BACE1 is necessary for experience-dependent homeostatic synaptic plasticity in visual cortex (Q33740069) (← links)
- Accumulation of intraneuronal Abeta correlates with ApoE4 genotype (Q33771171) (← links)
- Molecular mechanisms of neurodegeneration in Alzheimer's disease (Q33871297) (← links)
- β-Amyloid inhibits E-S potentiation through suppression of cannabinoid receptor 1-dependent synaptic disinhibition (Q33966489) (← links)
- Synapses, synaptic activity and intraneuronal abeta in Alzheimer's disease (Q34028039) (← links)
- Modeling Presenilin-Dependent Familial Alzheimer's Disease: Emphasis on Presenilin Substrate-Mediated Signaling and Synaptic Function (Q34078241) (← links)
- Reduced levels of the tyrosine phosphatase STEP block β amyloid-mediated GluA1/GluA2 receptor internalization (Q34212491) (← links)
- Oligomeric amyloid-{beta} inhibits the proteolytic conversion of brain-derived neurotrophic factor (BDNF), AMPA receptor trafficking, and classical conditioning (Q34251263) (← links)
- Amyloid-β pathology and APOE genotype modulate retinoid X receptor agonist activity in vivo. (Q34431008) (← links)
- Activation of D1/D5 dopamine receptors protects neurons from synapse dysfunction induced by amyloid-beta oligomers (Q34536809) (← links)
- Oligomeric Aβ-induced synaptic dysfunction in Alzheimer's disease (Q34544243) (← links)
- Amyloid beta impairs mitochondrial anterograde transport and degenerates synapses in Alzheimer's disease neurons (Q34590202) (← links)
- Drosophila models of neurodegenerative diseases (Q34610331) (← links)
- Phospho-dependent ubiquitination and degradation of PAR-1 regulates synaptic morphology and tau-mediated Aβ toxicity in Drosophila. (Q35018751) (← links)
- AMPA receptor activation promotes non-amyloidogenic amyloid precursor protein processing and suppresses neuronal amyloid-β production (Q35036124) (← links)
- Reduction of β-amyloid and γ-secretase by calorie restriction in female Tg2576 mice (Q35137488) (← links)
- Detection of peri-synaptic amyloid-β pyroglutamate aggregates in early stages of Alzheimer's disease and in AβPP transgenic mice using a novel monoclonal antibody (Q35151492) (← links)