Estudiante

Although cultural consumption continues to grow, arts institutions more than ever must focus on the needs of their clients. How does one reconcile cultural needs with the need for high attendance rates? Visitor loyalty seems to be the best strategy in response to this challenge. Having first touched on the attraction of loyalty, we attempt here to understand what motivates visitors (clients) to engage in a long-term relationship with a museum. A conceptual model is presented and illustrated with a case study: contemporary art work center. Aunque el consumo cultural continúa creciendo, las instituciones relacionadas con el mundo del arte deben centrarse más que nunca en las necesidades de sus clientes.áCámo se pueden compatibilizar las necesidades culturales con la necesidad de lograr altas tasas de visitantes? La lealtad de los visitantes parece ser en este orden de cosas la mejor estrategia de respuesta a la situatión planteada. Tras una breve referencia a la atracción y fidelización, con el presente trabajo se pretende entender mejor qué es lo que hace que los visitantes (clientes) se decidan a establecer una relatión a largo plazo con un museo. A tal fin, se propone un modelo conceptual ilustrado con el estudio de un caso referido a un centra de arte contemporáneo.

The noradrenergic innervation of the ovary of prepubertal rats causes an inhibitory response of the follicles to gonadotrophins, leading to ovulation. We investigated the possibility that noradrenergic peripheral denervation at birth, produced by treatment with guanethidine, modifies the positive feedback effects of gonadotrophins and oestradiol in prepubertal rats, and also the possibility that peripheral denervation can modify the anovulatory syndrome induced by androgenization at birth. Noradrenergic peripheral denervated rats of 18 days of age treated with pregnant mare serum gonadotrophin (PMSG) ovulated 96 h later, while normal animals did not ovulate (4/9 vs 0/12, P < 0.05) and the number of ova shed was lower than in rats which ovulated spontaneously at first vaginal oestrus (3.5 +/- 0.6 vs 8.3 +/- 0.4 (S.E.M.), P < 0.01). Oestradiol benzoate (10 micrograms) did not induce ovulation in either normal or denervated animals (0/11 and 0/11). The anovulatory syndrome induced by the administration of testosterone propionate (75 micrograms) at birth was partially blocked by noradrenergic peripheral denervation (4/7 ovulated vs 0/10). The results suggest that some neural information arising from the ovary modulates, in an inhibitory way, the stimulatory feedback mechanisms required to induce ovulation. Partial inhibition of the anovulatory syndrome resulting from androgenization caused by peripheral noradrenergic denervation suggests that noradrenergic neural information sent by the ovary to the hypothalamus results in a decreased concentration of noradrenaline in the hypothalamus and in the aromatization of androgens to oestrogens.