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Mx. of Acute Git Haem.

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MANAGEMENT OF ACUTE

GIT HAEMORRHAGE

G. JUMBI

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1. CAUSES
• 1. General causes (bleeding
disorders).
• 2. Local causes (see below).

Local Causes - Upper GIT


Haemorrhage.
• Peptic ulcers (Duodenal Ulcer and
gastric ulcers
50-75%
Gastric erosion (Haemorrhagic gastric
10% 2
2. CAUSES
Local Causes - Lower GIT
Haemorrhage.
• Diverticular disease
50%
• Haemorrhoids
• Colorectal carcinoma.
• Polyps.
• Colitis (e.g ulcerative colitis)
•  3
PATHOPHYSIOLOGY
• COVERT SHOCK.
• COMPENSATED SHOCK.
• DECOMPENSATED SHOCK.
Early decompensation.
Severe decompensation.
• UNRESPONSIVE SHOCK.
• IRREVERSIBLE SHOCK.
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HISTORY & PHYSICAL
EXAMINATION
UPPER GIT BLEEDING.
• HAEMATEMESIS.
• MALAENNA.
• PER RECTAL BLEEDING.
• SHOCK.
• Hx OF PUD.
• Hx OF NSAIDs.
• Hx OF MALLORY-WEISS SYNDROME.
• SIGNS OF PORTAL HPT.
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HISTORY & PHYSICAL
EXAMINATION
LOWER GIT BLEEDING.
• TORRENTIAL PER RECTAL
BLEEDING.
• SHOCK.
• PROCTOSCOPY.

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INVESTIGATIONS.
• BASELINE INVESTIGATIONS.
Hb, Hct, Group & X-Match,Coag.screen.
• ECG.
• ENDOSCOPY.
OGD.
Proctoscopy.
Sigmoidoscopy.
Colonoscopy.

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2. INVESTIGATIONS.
• BARIUM CONTRAST STUDIES.
Barium meal.
Barium enema (double contrast
studies).
• ANGIOGRAPHY/interventional
radiology).
Arteriography (coeliac & mesenteric).
Venography (portal vein, splenic vein, varices.

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TREATMENT.
• MEDICAL MANAGEMENT 50-75%.

• NON – OPERATIVE HAEMOSTASIS


HAEMOSTASIS INTERVENTIONS.

• SURGICAL INTERVENTIONS.

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1. MEDICAL MANAGEMENT.
RESUSCITATION (Shocked patients).
AIRWAY.
BREATHING.
CICULATION ( CARDIAC ARREST
& MANAGEMENT OF SHOCK).

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2. MEDICAL MANAGEMENT.
MANAGEMENT OF HAMORRHAGIC
SHOCK.
• THE SHOCK POSITION.
• DRUGS (O2, NaHCO3, HCT).
• GP & X-MATCH.
• I.V. LINE
• BLOOD REPLACEMENT.
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CLINICAL CORRELATION
Clinical correlation % of Blood (For a 70 Kg Blood
(Severity of shock) lost (Adult Required

<10%-15% <750 ml No blood


. Shock absent (covert shock): PR = 60-100/min required
BP=110-140;

bsent vasoconstriction(VC);

rine output (UO) = 40 – 50 ml/hr.

2. Compensated Shock: PR = 100-120/ml 10% - 30% 750ml— ½L–L


BP = 110-140;VC = +; UO =30-40 ml/hr. 11/2L

30 – 50% 1½l–2½L 1
. Early decompensation: PR = 120-140

P = 70-110; VC = ++;UO = <30ml/hr

4. Severe decompensation: PR =>140/min > 50% 2½-3½ > 3L


BP = 0-70; VC = +++; UO = Zero
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3. MEDICAL MANAGEMENT.
MONITORING OF SHOCK.
• CLINICAL MONITORING.
• URINARY OUTPUT.
• NGT / RECTAL TUBE MONITORING.
• BLOOD GAS ANALYSIS
• CVP LINE.
• LVFP (FCT, PAWP).
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OUTCOME OF MEDICAL
TREATMENT.
• BLEEDING STOPS 50-75%.
OGD / BA. STUDIES AFTER 24
HOURS.
• BLEEDING FAILS TO STOP 25-50%
1. NON-OPERATIVE
INTERVENTION.
2. SURGICAL INTERVENTION.

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NON – OPERATIVE
INTERVENTIONS.
• EMERGENCY ENDOSCOPY (UPPER GIT
ONLY). (Coagulation, sclerotherapy,
polypectomy).

• TAMPONADE (VARICES).

• INTERVEVNTIONAL RADIOLOGY:
ARTERIAL EMBOLIZATION.
VENOUS EMBOLIZATION.
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SURGICAL INTERVENTION.
(LAPAROTOMY).
Duodenal ulcer–ligation of the bleeder +
Vagotomy &
Drainage.
Gastric ulcer – Ligation of bleeder +
Partial gastrectomy.
Mallory–Weiss – Gastrotomy, ligation of
the bleeder +
repair.
Oesophageal varices –Transection of
oesophagus
(or the
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stomach).
2.SURGICAL INTERVENTION
• Ca. stomach - Gastrectomy (total or partial).
• Diverticular disease- Segmental colectomy
+ colostomy.
• C.A. colon – Partial colectomy +
colostomy.
• Polyps - Colotomy + polypectomy.

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