Hypocalcaemia

Dental disease is considered as one of the, if not, the most common disorders seen in pet rabbits. This article provides a review of the scientific literature and an overview of the peculiarities of calcium homeostasis in the rabbit in an attempt to draw together current thinking on the cause of dental disease. A complete understanding of the aetiology and pathophysiology of rabbit dental disease is necessary for the veterinary practitioner to establish a proper therapeutic plan, prognosis and ultimately prevention of this common cause of morbidity and mortality in pet rabbits.

Context. Diabetes mellitus is the most frequent chronic complication in pregnancy and continues to contribute to increased perinatal morbidity and mortality in newborns. Macrosomia, respiratory distress syndrome, metabolic and electrolytic disturbances, and increased rates of congenital structural defects are well-known neonatal complications associated with maternal diabetes, even if well-controlled. Case report. A macrosomic infant born from an insulin-dependent mother, with uncontrolled diabetes and lack of adequate prenatal care, prenatally diagnosed with hydrocephaly showed a complicated postnatal course. Initial respiratory distress syndrome and transient hypoglycemia, rapidly corrected under treatment, were followed by persistent hypocalcemia and hyperphosphatemia due to hypoparathyroidism and evolving hydrocephaly. Ventriculoperitoneal shunting was followed by resolution of hypocalcemia, but seizures associated with schizencephaly and recurrent respiratory tract infections, aggravated by spondylocostal dysplasia, concurred to infant's demise at the age of 5 months. Conclusions. The reported case is rare due to multiple aspects: persistent hypoparathyroidism, uncommon association of schizencephaly, and even rarely association with spondylocostal dysplasia, all these conditions requiring a multidisciplinary therapeutic approach. Also, the reported case is evocative for challenges associated with infants born from diabetic mothers.

Present communication about the doe which was brought to clinic with the complaint of prolapse of uterus. Upon clinical examination it had soiled, necrosed debris on the uterus with abnormal vital parameters. On laboratory examination doe had anaemia, leucocytosis, low levels of total serum protein, serum albumin and calcium levels. Doe also had severe Haemonchus spp. infection. The everted organ was washed with KMnO4 solution and gross debris was removed. Epidural anesthesia was achieved by 2 % lignocaine hydrochloride and prolapsed mass was slowly pushed inside the pelvic cavity through vagina and placed in its normal position. Doe was treated by administering the enrofloxacin, tolfenamic acid, oxytocin, calcium borogluconate and fenbendazole. After close monitoring for three days no incidence of prolapse was noticed.
Key Words: Doe, Haemonchus, Hypocalcaemia, Treatment, Uterine prolapse