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I Human Case - Edited

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Running head: I HUMAN CASE 1

I Human Case: Left Facial Paralysis

Student’s Name

Institutional Affiliation

Instructor

Date
I HUMAN CASE 2

I Human Case: Left Facial Paralysis

Facial paralysis is a common presentation that could be an implication in a wide variety

of pathological conditions. As such, making a diagnosis of such a presentation could be difficult.

However, it imperative that a clinician makes an accurate diagnosis to inform the management of

the underlying condition for better outcomes (Kestler et al., 2017). Achieving such an objective

would call for the collection of a detailed history, a thorough physical examination, objective

diagnostic testing, and informed management. As such, this management takes note of the case

of a 41-year-old patient to discuss how a detailed history, physical examination, and diagnostic

testing could inform the diagnosis of infective endocarditis for proper management.

History of Presenting Illness

A 41-year-old male patient presents with a 2-week history of complaints of left facial

paralysis, acute on the onset. The patient also reports a history of persistent cough, fever, chills,

and different new lesions on the skin that also started about a fortnight ago. The fever was high

grade, intermittent, and not associated with rigors (Papadakis, McPhee, & Rabow, 2014).

However, the patient reports no history of seizure; bladder or bowel incontinence; food

regurgitation; burning sensation on urination; increased urinary frequency; abdominal pain,

nausea, and vomiting; or cough with expectoration and breathlessness. The patient denies any

history of similar episodes but reports to have had been managed for a cardiovascular lesion

about a year ago for which prescription drugs were administered. However, the patient denies

any history of alcohol consumption nor cigarette smoking with no family history of

cardiovascular pathology.

Physical Examination
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On examination, the vitals reveal that the patient is febrile with an oral temperature of

101oF, a pulse rate of 136 beats per minute revealing tachycardia, a blood pressure of 168/78

showing systolic hypertension, and a respiratory rate of 22 breaths per minute revealing

tachypnea. Cardiovascular examination reveals a new onset of 4/6 systolic ejection murmur in all

the valve areas, but loudest at the apex, which locates at the midclavicular line fifth intercostal

space. Of note on dermatological assessment is the presence of multiple macular lesions on the

skin evident bilaterally on the distal extremities (Papadakis, McPhee, & Rabow, 2014). On the

right arm, as well as on the back, plaques of psoriasis are evident with plaques on healing stages

visible not only on the arms but also on the hands. There is also the presence of splinter

hemorrhages bilaterally on the extremities. On the central nervous system examination, the

patient is conscious and oriented both in time and space. However, despite having intact

memory, the patient is dysarthria with anomic aphasia (Jameson et al., 2017). Cranial nerve

assessment reveals upper motor neuronal lesions on the facial nerve on the left side.

Differential Diagnoses and Laboratory Tests

From the assessment, the differential diagnoses include tuberculosis, Bell’s palsy, mitral

valve regurgitation, endocarditis, and transient ischemic attack. To rule out the differential

diagnoses and come up with the definitive diagnosis, the tests ordered included echocardiogram,

two sets of blood culture, complete blood count, BMP, both PA and lateral chest X-ray, and

brain MRI. Of the two samples of blood taken for culture, one was negative, whereas the other

depicted coagulase-negative staphylococci. A repeat of blood culture after five days also

revealed coagulase-negative staphylococci (Ross et al., 2017). An echocardiogram reveals

moderate mitral regurgitation, positive valvar vegetation, and ruptured chordae tendinae. As

such, brain MRI revealed small infarcts on the caudal aspect of the right paracentral gyrus with
I HUMAN CASE 4

an elevation of WBC to 20 x 109/L. From the history, assessment findings, and test results, the

definitive diagnosis as bacterial endocarditis.

Management Plan

As such, the management plan for this patient would aim at fulfilling such goals as

eradicating the underlying infectious agent from the vegetation besides addressing the

complications that would come in association with infective endocarditis, including both cardiac

and extracardiac consequences. As such, the management of this patient would employ

vancomycin 1 g IV BID for 4 weeks and gentamycin 80 mg BID for five days (Jameson et al.,

2017). However, it would be viable to continue weekly echocardiogram to assess the reduction in

the size of the vegetation with a follow-up with blood culture after three weeks to monitor the

progress of treatment.

Conclusion

In conclusion, this management takes note of the case of a 41-year-old patient to discuss

how a detailed history, physical examination, and diagnostic testing could inform the diagnosis

of infective endocarditis for proper management. The 41-year-old male patient presents with a 2-

week history of complaints of complaints of left facial paralysis, acute on onset. On examination,

the vitals reveal that the patient is febrile with an oral temperature of 101oF, a pulse rate of 136

beats per minute revealing tachycardia, a blood pressure of 168/78 revealing systolic

hypertension, and a respiratory rate of 22 breaths per minute revealing tachypnea. The

management plan for this patient would aim at fulfilling such goals as eradicating the underlying

infectious agent from the vegetation besides addressing the complications that would come in

association with infective endocarditis.


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References

Jameson, J. L., Fauci, A. S., Kasper, D. L., Hauser, S. L., Longo, D. L., & Loscalzo, J. (2017).

Harrison's Principles of Internal Medicine 19th Edition and Harrison's Manual of

Medicine 19th Edition (EBook) VAL PAK. McGraw Hill Professional.

Kestler, M., Munoz, P., Marin, M., Goenaga, M. A., Viedma, P. I., de Alarcón, A., ... & Costas,

C. (2017). Endocarditis caused by anaerobic bacteria. Anaerobe, 47, 33-38.

Liesenborghs, L., Meyers, S., Lox, M., Criel, M., Claes, J., Peetermans, M., ... & Missiakas, D.

(2019). Staphylococcus aureus endocarditis: distinct mechanisms of bacterial adhesion to

damaged and inflamed heart valves. European heart journal, 40(39), 3248-3259.

Papadakis, M.A., McPhee, S.J., & Rabow, M.W. (2014). Current medical diagnosis &

treatment: Geriatric (2nd ed.). New York, NY: Routledge.

Ross, K. M., Mehr, J. S., Greeley, R. D., Montoya, L. A., Kulkarni, P. A., Frontin, S., ... &

Montana, B. E. (2018). Outbreak of bacterial endocarditis associated with an oral surgery

practice: New Jersey public health surveillance, 2013 to 2014. The Journal of the

American Dental Association, 149(3), 191-201.

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